Evidence map›Paper›PMID 37316687›Full record

ReviewImmunologic research2023

COVID-19 infection: a possible induction factor for development of autoimmune diseases?

Nikoo Saeedi, Narjes Sadat Farizani Gohari, Amir Ali Moodi Ghalibaf, Ali Dehghan, Mohammad Bagher Owlia

Abstract readReview
PubMed Publisher
In one paragraph

Review in Immunologic research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed, 1 pooled it
1.7field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 1 synthesis or guideline pooled it, 9 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Article
  4. Article
  5. Article
  6. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Nikoo Saeedi *Student Research Committee, Islamic Azad University, Mashhad Branch, Mashhad, Iran. nikoosaeedie@gmail.com.ORCID 0000-0003-1309-4408
Narjes Sadat Farizani Gohari *Interest Group of CoronaVirus 2019 (IGCV-19), Universal Scientific Education and Research Network (USERN), Yazd, Iran.
Amir Ali Moodi GhalibafStudent Research Committee, Faculty of Medicine, Birjand University of Medical Sciences, Birjand, Iran.
Ali DehghanDivision of Rheumatology, Shahid Sadoughi University of Medical Sciences, Yazd, Iran.
Mohammad Bagher OwliaDivision of Rheumatology, Shahid Sadoughi University of Medical Sciences, Yazd, Iran.
Shahid Sadoughi University of Medical Sciences and Health Services · IRIslamic Azad University, Mashhad · IRUniversal Scientific Education and Research Network · IRYazd University · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Following the global spread of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, the importance of investigation of the pathogenesis and immunological characteristics of COVID-19 became quite clear. Currently, there are reports indicating that COVID-19 is able to induce autoimmune responses. Abnormal immune reactions are a cornerstone in the pathogenicity of both conditions. Detection of autoantibodies in COVID-19 patients may suggest a link between COVID-19 and autoimmunity. In this study, we focused on the similarities and possible differences between COVID-19 and autoimmune disorders to explore the relationship between them. Comparing the pathogenicity of SARS-CoV-2 infection with autoimmune conditions revealed significant immunological properties of COVID-19 including the presence of several autoantibodies, autoimmunity-related cytokines, and cellular activities that could be useful in future clinical studies aiming at managing this pandemic.

Indexed as

Autoimmune DiseasesCOVID-19AutoantibodiesAutoimmunityHumansSARS-CoV-2AutoantibodiesAutoimmune diseasesCOVID-19CytokinesPathogenesis

Identifiers

PMID37316687
OpenAlexW4380569240

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.