Evidence map›Paper›PMID 37294500›Full record

ArticleTranslational stroke research2024

Neutrophil-activating Peptide 2 as a Novel Modulator of Fibrin Clot Properties in Patients with Atrial Fibrillation.

Michał Ząbczyk, Joanna Natorska, Paweł T Matusik, Patrycja Mołek, Wiktoria Wojciechowska, Marek Rajzer, Renata Rajtar-Salwa, Tomasz Tokarek, Aleksandra Lenart-Migdalska, Maria Olszowska and 1 more

Open access · hybridAbstract read
In one paragraph

Article in Translational stroke research, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.9field-weighted citation impact, top 24% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 4 citations in OpenAlex.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 2 institutions in 1 country.

Michał ZąbczykDepartment of Thromboembolic Disorders, Institute of Cardiology, Jagiellonian University Medical College, Pradnicka 80, 31-202, Krakow, Poland.
Joanna NatorskaDepartment of Thromboembolic Disorders, Institute of Cardiology, Jagiellonian University Medical College, Pradnicka 80, 31-202, Krakow, Poland.
Paweł T MatusikInstitute of Cardiology, Faculty of Medicine, Jagiellonian University Medical College, Pradnicka 80, Kraków, Poland.
Patrycja MołekKrakow Centre for Medical Research and Technologies, the John Paul II Hospital, Pradnicka 80, Krakow, Poland.
Wiktoria Wojciechowska1st Department of Cardiology, Interventional Electrocardiology and Arterial Hypertension, Jagiellonian University Medical College, Jakubowskiego 2, Kraków, Poland.
Marek Rajzer1st Department of Cardiology, Interventional Electrocardiology and Arterial Hypertension, Jagiellonian University Medical College, Jakubowskiego 2, Kraków, Poland.
Renata Rajtar-SalwaDepartment of Cardiology and Cardiovascular Interventions, University Hospital, Jakubowskiego 2, Krakow, Poland.
Tomasz TokarekCenter for Invasive Cardiology, Electrotherapy and Angiology, Kilinskiego 68, Nowy Sacz, Poland.
Aleksandra Lenart-MigdalskaDepartment of Cardiac and Vascular Diseases, Faculty of Medicine, Institute of Cardiology, Jagiellonian University Medical College, John Paul II Hospital, Pradnicka 80, Kraków, Poland.
Maria OlszowskaDepartment of Cardiac and Vascular Diseases, Faculty of Medicine, Institute of Cardiology, Jagiellonian University Medical College, John Paul II Hospital, Pradnicka 80, Kraków, Poland.
Anetta UndasDepartment of Thromboembolic Disorders, Institute of Cardiology, Jagiellonian University Medical College, Pradnicka 80, 31-202, Krakow, Poland. mmundas@cyf-kr.edu.pl.ORCID 0000-0002-0667-8109
Jagiellonian University · PLJohn Paul II Hospital · PL

Funding

Uniwersytet Jagielloński Collegium Medicum N41/DBS/000906
6 · The paper itself

Abstract

Neutrophil-activating peptide 2 (NAP-2, CXCL7), a platelet-derived neutrophil chemoattractant, is involved in inflammation. We investigated associations between NAP-2 levels, neutrophil extracellular traps (NETs) formation, and fibrin clot properties in atrial fibrillation (AF). We recruited 237 consecutive patients with AF (mean age, 68 ± 11 years; median CHA

Indexed as

Atrial FibrillationFibrinAgedAged, 80 and overExtracellular TrapsFemaleHumansLeukocyte ElastaseMaleMiddle AgedNeutrophilsELANE protein, humanFibrinLeukocyte ElastaseAtrial fibrillationFibrin clotNeutrophil-activating peptide 2Oxidative stress

Identifiers

PMID37294500
PMCPMC10250863
OpenAlexW4379967141

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.