Evidence map›Paper›PMID 37264959›Full record

ArticleInternational journal of oncology2023

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Wenjun Liu, Zilang Zhang, Xitu Luo, Kai Qian, Baojun Huang, Jianmin Liang, Zhihao Ma, Jianzhong Deng, Chengyu Yang

Open access · hybridAbstract read
In one paragraph

Article in International journal of oncology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
5.5field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Review
  3. The Emerging Role of METTL3 in Lung Diseases.International journal of molecular sciences · 2025
    Review
  4. Review
  5. Review
  6. Article
  7. Review
  8. AmiCa: Atlas of miRNA-gene correlations in cancer.Computational and structural biotechnology journal · 2024
    Article
  9. Article
  10. Article
  11. The essential roles of lncRNAs/PI3K/AKT axis in gastrointestinal tumors.Frontiers in cell and developmental biology · 2024
    Review
  12. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 4 institutions in 1 country.

Wenjun Liu *The First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Zilang Zhang *Department of AnoRectal Surgery, The First People's Hospital of Foshan, Foshan, Guangdong 528010, P.R. China.
Xitu LuoThe First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Kai QianDivision of Vascular and Interventional Radiology, Department of General Surgery, Nanfang Hospital, Southern Medical University, Guangzhou, Guangdong 510000, P.R. China.
Baojun HuangThe First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Jianmin LiangThe First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Zhihao MaThe First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Jianzhong DengDepartment of AnoRectal Surgery, The First People's Hospital of Foshan, Foshan, Guangdong 528010, P.R. China.
Chengyu YangThe First Department of General Surgery, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, Guangdong 510150, P.R. China.
Third Affiliated Hospital of Guangzhou Medical University · CNFirst People's Hospital of Foshan · CNGuangzhou Medical University · CNSouthern Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Long noncoding RNAs (lncRNAs) are a type of regulatory molecule with potential roles in the development of several different malignancies. However, the underlying mechanisms of lncRNAs in colorectal cancer (CRC) are incompletely understood. The present study investigated the molecular mechanism of LINC02038 in CRC. LINC02038 expression was decreased in CRC tissues compared to the para‑cancerous tissues and LINC02038 overexpression markedly reduced the proliferation, vitality, migration and invasive ability and greatly accelerated apoptosis of colorectal cancer cells. Bioinformatics examination indicated that LINC02038 may have targeted microRNA (miR)‑552‑5p. RNA immunoprecipitation and luciferase reporter assays showed that LINC02038 served as a sponge for miR‑552‑5p, hindering target gene FAM172A of miR‑552‑5p degradation. Moreover, methylated RNA immunoprecipitation (MeRIP)‑qualitative PCR assays revealed that YTHDF2 could identify and regulate the METTL3‑mediated LINC02038 N6‑methyladenosine (m

Indexed as

Colorectal NeoplasmsMicroRNAsRNA, Long NoncodingBinding, CompetitiveCell Line, TumorCell MovementCell ProliferationGene Expression Regulation, NeoplasticHumansMethyltransferasesPhosphatidylinositol 3-KinasesProteinsProto-Oncogene Proteins c-aktARB2A protein, humanMethyltransferasesMETTL3 protein, humanMicroRNAsMIRN552 microRNA, humanPhosphatidylinositol 3-KinasesProteinsProto-Oncogene Proteins c-aktRNA, Long NoncodingceRNAcolorectal cancerLINC02038N6‑methyladenosineprogression

Identifiers

PMID37264959
PMCPMC10552712
OpenAlexW4379094014

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.