Evidence map›Paper›PMID 37254185›Full record

ReviewCell communication and signaling : CCS2023

Cellular crosstalk in atherosclerotic plaque microenvironment.

Elmira Mahdinia, Nafiseh Shokri, Abdolkarim Talebi Taheri, Sahar Asgharzadeh, Mohammad Elahimanesh, Mohammad Najafi

Abstract readVideo-Audio MediaReview
In one paragraph

Review in Cell communication and signaling : CCS, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 22 papers.

0numbers the graph read from it
0cells of the map it votes in
22citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

22 citing papers in PubMed.

  1. Ilexoside K Ameliorates Atherosclerosis by Suppressing CD72Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
    Article
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  11. Single-Cell RNA Sequencing Reveals Granzyme KJournal of the American Heart Association · 2025
    Article
  12. Mitigating atherosclerosis: Integrating vaccines with gene targets.American heart journal plus : cardiology research and practice · 2025
    Review
  13. Article
  14. Article
  15. Article
  16. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Elmira Mahdinia *Department of Clinical Biochemistry, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Nafiseh Shokri *Department of Clinical Biochemistry, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Abdolkarim Talebi TaheriDepartment of Clinical Biochemistry, Faculty of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
Sahar AsgharzadehDepartment of Clinical Biochemistry, Faculty of Medicine, Ghazvin University of Medical Sciences, Ghazvin, Iran.
Mohammad ElahimaneshDepartment of Clinical Biochemistry, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.
Mohammad NajafiDepartment of Clinical Biochemistry, School of Medicine, Iran University of Medical Sciences, Tehran, Iran. nbsmmsbn@iums.ac.ir.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Atherosclerosis is an underlying pathology of many vascular diseases as a result of cellular, structural and molecular dysfunctions within the sub-endothelial space. This review deals with the events involved in the formation, growth and remodeling of plaque, including the cell recruitment, cell polarization, and cell fat droplets. It also describes cross talking between endothelial cells, macrophages, and vascular smooth muscle cells, as well as the cellular pathways involved in plaque development in the plaque microenvironment. Finally, it describes the plaque structural components and the role of factors involved in the rupture and erosion of plaques in the vessel. Video Abstract.

Indexed as

AtherosclerosisPlaque, AtheroscleroticEndothelial CellsHumansAtherosclerosisErosionPlaquePolarizationRupture

Identifiers

PMID37254185
PMCPMC10227997

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.