Evidence map›Paper›PMID 37234927›Full record

ArticleJournal of the ASEAN Federation of Endocrine Societies2023

Bisphenol S Increases Cell Number and Stimulates Migration of Endometrial Epithelial Cells.

Kimberly Benjamin, Cielo Mae Marquez, Madeleine Morta, Emmanuel Marc Reyes, Lemnuel Aragones, Michael Velarde

Open access · diamondAbstract read
In one paragraph

Article in Journal of the ASEAN Federation of Endocrine Societies, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.6field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Kimberly BenjaminDepartment of Biology, College of Arts and Sciences, University of the Philippines Manila.
Cielo Mae MarquezInstitute of Biology, College of Science, University of the Philippines Diliman, Quezon City, Philippines.
Madeleine MortaInstitute of Biology, College of Science, University of the Philippines Diliman, Quezon City, Philippines.
Emmanuel Marc ReyesInstitute of Biology, College of Science, University of the Philippines Diliman, Quezon City, Philippines.
Lemnuel AragonesInstitute of Environmental Science and Meteorology, College of Science, University of the Philippines Diliman, Quezon City, Philippines.
Michael VelardeInstitute of Biology, College of Science, University of the Philippines Diliman, Quezon City, Philippines.
University of the Philippines Diliman · PHUniversity of the Philippines Manila · PH

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Objective: To determine whether bisphenol S (BPS), a common substitute for bisphenol A (BPA), induces cell proliferation and migration in human endometrial epithelial cells (Ishikawa) and adult mouse uterine tissues. Methodology: Human endometrial Ishikawa cells were exposed to low doses of BPS (1 nM and 100 nM) for 72 hours. Cell proliferation was assessed through the viability assays MTT and CellTiter-Glo Results: BPS increased cell number and stimulated migration in Ishikawa cells, in association with the upregulation of estrogen receptor beta Conclusion: Overall,

Indexed as

EndometriumUterusAnimalsBisphenol A CompoundsBisphenol S CompoundsCell CountCell LineEpithelial CellsFemaleHumansMicebisphenol ABisphenol A Compoundsbisphenol SBisphenol S CompoundsBPSendocrine-disrupting chemicalshyperplasiaIshikawa cellsuterus

Identifiers

PMID37234927
PMCPMC10207871
OpenAlexW4312997965

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.