Evidence map›Paper›PMID 37213054›Full record

ReviewCurrent nutrition reports2023

Pathophysiology and Nutritional Approaches in Polycystic Ovary Syndrome (PCOS): A Comprehensive Review.

M Di Lorenzo, N Cacciapuoti, M S Lonardo, G Nasti, C Gautiero, A Belfiore, B Guida, M Chiurazzi

Open access · hybridAbstract readReview
In one paragraph

Review in Current nutrition reports, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 67 papers, 7 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
67citing papers in PubMed, 7 pooled it
20.5field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

67 citing papers in PubMed, 7 syntheses or guidelines pooled it, 75 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Pooled it
  4. Pooled it
  5. Pooled it
  6. Pooled it
  7. Pooled it
  8. Trial
  9. Trial
  10. Trial
  11. Trial
  12. Article
  13. Article
  14. Review
  15. Review
  16. Article
  17. Review
  18. Article
  19. Article
  20. Review

7 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

M Di Lorenzo *Department of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
N Cacciapuoti *Department of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
M S LonardoDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
G NastiDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
C GautieroDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
A BelfioreDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
B GuidaDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy.
M ChiurazziDepartment of Clinical Medicine and Surgery, University of Naples "Federico II", Naples, Italy. martina.chiurazzi88@gmail.com.ORCID 0000-0003-2406-890X
University of Naples Federico II · ITOspedale Antonio Cardarelli · ITOspedale D. Cotugno · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of reviewPolycystic ovary syndrome (PCOS) is the most common endocrine and metabolic disorder in women of reproductive age worldwide. This disease causes menstrual, metabolic, and biochemical abnormalities such as hyperandrogenism, oligo-anovulatory menstrual cycles, polycystic ovary, hyperleptinemia, insulin resistance (IR), and cardiometabolic disorders, often associated with overweight or obesity and visceral adiposity. RECENT

findingsThe etiology and pathophysiology of PCOS are not yet fully understood, but insulin seems to play a key role in this disease. PCOS shares an inflammatory state with other chronic diseases such as obesity, type II diabetes, and cardiovascular diseases; however, recent studies have shown that a healthy nutritional approach can improve IR and metabolic and reproductive functions, representing a valid therapeutic strategy to ameliorate PCOS symptomatology. This review aimed to summarize and collect evidence about different nutritional approaches such as the Mediterranean diet (MedDiet) and the ketogenic diet (KD), as well as bariatric surgery and nutraceutical supplementation as probiotics, prebiotics, and synbiotics, among the others, used in patients with PCOS.

Indexed as

Diabetes Mellitus, Type 2HyperandrogenismInsulin ResistancePolycystic Ovary SyndromeFemaleHumansObesityHormone profileInsulin resistanceNutritional approachesPolycystic ovary syndrome

Identifiers

PMID37213054
PMCPMC10444658
OpenAlexW4377220378

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.