Evidence map›Paper›PMID 37204635›Full record

ReviewReproductive sciences (Thousand Oaks, Calif.)2023

The Roles of Autophagy in the Genesis and Development of Polycystic Ovary Syndrome.

Di Cheng, Biao Zheng, Ying Sheng, Zhaoming Zeng, Zhongcheng Mo

Abstract readReview
PubMed Publisher
In one paragraph

Review in Reproductive sciences (Thousand Oaks, Calif.), 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.7field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Review
  3. Review
  4. Article
  5. Article
  6. Review
  7. Review
  8. Article
  9. Article
  10. Article
  11. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Di ChengGuangxi Key Laboratory of Diabetic Systems Medicine, Department of Histology and Embryology, Guilin Medical University, Guangxi, 541199, Guilin, China.
Biao ZhengGuangxi Key Laboratory of Diabetic Systems Medicine, Department of Histology and Embryology, Guilin Medical University, Guangxi, 541199, Guilin, China.
Ying ShengDepartment of Obstetrics, The First Affiliated Hospital of University of South China, Hengyang, 421001, Hunan, China.
Zhaoming ZengJoint Laboratory of Chronic Disease Prevention and Research, Guilin Medical University, Hunan Mingshun Pharmaceutical Co., Ltd, Shaodong, Hunan, 422800, Guilin, China. 381410765@qq.com.
Zhongcheng MoGuangxi Key Laboratory of Diabetic Systems Medicine, Department of Histology and Embryology, Guilin Medical University, Guangxi, 541199, Guilin, China. zhchmo@glmc.edu.cn.ORCID http://orcid.org/0000-0002-0706-9364
Guilin Medical University · CNFirst Affiliated Hospital of University of South China · CN

Funding

Guangxi Natural Science Foundation 2020GXNSFAA238008Scientific research project of Hunan Health Committee 202105012226the Horizontal Cooperation project with Hunan Mingshun Pharmaceutical Co., Ltd 2021GLHX01
6 · The paper itself

Abstract

Polycystic ovary syndrome (PCOS) is a common and frequent disease and always leads endocrine and metabolic disorder among women in reproductive age. Ovary is the main organ involved in polycystic ovary syndrome, and its function impairment will lead to reproductive dysfunction. Some recent studies have demonstrated that autophagy plays an important role in the pathogenesis of PCOS, and there are many different mechanisms that affect autophagy and the occurrence of PCOS, and they provide a new direction for us to predict the mechanism of PCOS. In this review, we discuss the role of autophagy in different ovarian cells: granulosa cells, oocytes, and theca cells, and introduce the important role that they play in the progress of PCOS. The main purpose of this review is to provide the research background and some relevant suggestions for our future work in autophagy and help us better explore the pathogenesis and autophagy mechanisms of PCOS. Furthermore, it will help us gain a new insight of the pathophysiology and treatment of PCOS.

Indexed as

Polycystic Ovary SyndromeAutophagyFemaleGranulosa CellsHumansOocytesAutophagyGranulosa cellOocytePCOSTheca cell

Identifiers

PMID37204635
OpenAlexW4377091816

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.