Evidence map›Paper›PMID 37182098›Full record

ArticleiScience2023

Cytotoxic CNS-associated T cells drive axon degeneration by targeting perturbed oligodendrocytes in

Tassnim Abdelwahab, David Stadler, Konrad Knöpper, Panagiota Arampatzi, Antoine-Emmanuel Saliba, Wolfgang Kastenmüller, Rudolf Martini, Janos Groh

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in iScience, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.9field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 7 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
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  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Tassnim AbdelwahabDepartment of Neurology, Section of Developmental Neurobiology, University Hospital Würzburg, Würzburg, Germany.
David StadlerDepartment of Neurology, Section of Developmental Neurobiology, University Hospital Würzburg, Würzburg, Germany.
Konrad KnöpperInstitute for Systems Immunology, University of Würzburg, Würzburg, Germany.
Panagiota ArampatziCore Unit Systems Medicine, University of Würzburg, Würzburg, Germany.
Antoine-Emmanuel SalibaHelmholtz Institute for RNA-based Infection Research, Helmholtz-Center for Infection Research, Würzburg, Germany.
Wolfgang KastenmüllerInstitute for Systems Immunology, University of Würzburg, Würzburg, Germany.
Rudolf MartiniDepartment of Neurology, Section of Developmental Neurobiology, University Hospital Würzburg, Würzburg, Germany.
Janos GrohDepartment of Neurology, Section of Developmental Neurobiology, University Hospital Würzburg, Würzburg, Germany.
Universitätsklinikum Würzburg · DEUniversity of Würzburg · DEHelmholtz Institute for RNA-based Infection Research · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Myelin defects lead to neurological dysfunction in various diseases and in normal aging. Chronic neuroinflammation often contributes to axon-myelin damage in these conditions and can be initiated and/or sustained by perturbed myelinating glia. We have previously shown that distinct

Indexed as

Components of the immune systemModel organismMolecular neuroscience

Identifiers

PMID37182098
PMCPMC10172788
OpenAlexW4366401392

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.