Evidence map›Paper›PMID 37130947›Full record

ReviewNature reviews. Endocrinology2023

Metformin: update on mechanisms of action and repurposing potential.

Marc Foretz, Bruno Guigas, Benoit Viollet

Registry-linked trialAbstract readReview
In one paragraph

Review in Nature reviews. Endocrinology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT07760883 (Effect of Metformin in Individuals With Type 2 Diabetes According to SLC16A11 Risk Variant Carrier Status), which is not on this map. Cited by 380 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
380citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT07760883 recruitingnot on this map

Effect of Metformin in Individuals With Type 2 Diabetes According to SLC16A11 Risk Variant Carrier Status

TypeobservationalSponsorInstituto Nacional de Ciencias Medicas y Nutricion Salvador ZubiranRan2020 to 2027Enrolled154ConditionsDiabetes Type 2
3 · Its place in the literature

Who cites it

380 citing papers in PubMed, 1 synthesis or guideline pooled it.

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320 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Marc ForetzUniversité Paris Cité, CNRS, Inserm, Institut Cochin, Paris, France.ORCID 0000-0001-7017-9032
Bruno GuigasDepartment of Parasitology, Leiden University Medical Center, Leiden, Netherlands.ORCID 0000-0002-8856-5799
Benoit ViolletUniversité Paris Cité, CNRS, Inserm, Institut Cochin, Paris, France. benoit.viollet@inserm.fr.ORCID 0000-0002-0121-0224

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Currently, metformin is the first-line medication to treat type 2 diabetes mellitus (T2DM) in most guidelines and is used daily by >200 million patients. Surprisingly, the mechanisms underlying its therapeutic action are complex and are still not fully understood. Early evidence highlighted the liver as the major organ involved in the effect of metformin on reducing blood levels of glucose. However, increasing evidence points towards other sites of action that might also have an important role, including the gastrointestinal tract, the gut microbial communities and the tissue-resident immune cells. At the molecular level, it seems that the mechanisms of action vary depending on the dose of metformin used and duration of treatment. Initial studies have shown that metformin targets hepatic mitochondria; however, the identification of a novel target at low concentrations of metformin at the lysosome surface might reveal a new mechanism of action. Based on the efficacy and safety records in T2DM, attention has been given to the repurposing of metformin as part of adjunct therapy for the treatment of cancer, age-related diseases, inflammatory diseases and COVID-19. In this Review, we highlight the latest advances in our understanding of the mechanisms of action of metformin and discuss potential emerging novel therapeutic uses.

Indexed as

COVID-19Diabetes Mellitus, Type 2MetforminGlucoseHumansHypoglycemic AgentsGlucoseHypoglycemic AgentsMetformin

Identifiers

PMID37130947
PMCPMC10153049

What OpenQuestion holds

Texttitle and abstract
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.