Evidence map›Paper›PMID 37096311›Full record

ArticleJournal of Korean medical science2023

Differential Expression of

Ji Hea Yu, Yun Ji Jung, Myung-Sun Kim, Sung-Rae Cho, Young-Han Kim

Open access · goldAbstract read
In one paragraph

Article in Journal of Korean medical science, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.3field-weighted citation impact, top 40% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 1 citations in OpenAlex.

  1. Review
  2. TGFBR2 coordinates the endometrial response to estrogen, regulating endometrial hyperplasia and fertility.Proceedings of the National Academy of Sciences of the United States of America · 2025
    Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Ji Hea YuDivision of Maternal Fetal Medicine, Department of Obstetrics and Gynecology, Severance Hospital, Yonsei University Medical College, Seoul, Korea.ORCID https://orcid.org/0000-0001-5686-9213
Yun Ji JungDivision of Maternal Fetal Medicine, Department of Obstetrics and Gynecology, Severance Hospital, Yonsei University Medical College, Seoul, Korea.ORCID https://orcid.org/0000-0001-6615-6401
Myung-Sun KimDepartment of Rehabilitation Medicine and Research Institute of Rehabilitation Medicine, Severance Hospital, Yonsei University Medical College, Seoul, Korea.ORCID https://orcid.org/0000-0002-9667-9964
Sung-Rae ChoDepartment of Rehabilitation Medicine and Research Institute of Rehabilitation Medicine, Severance Hospital, Yonsei University Medical College, Seoul, Korea. SRCHO918@yuhs.ac.ORCID https://orcid.org/0000-0003-1429-2684
Young-Han KimDivision of Maternal Fetal Medicine, Department of Obstetrics and Gynecology, Severance Hospital, Yonsei University Medical College, Seoul, Korea. YHKIM522@yuhs.ac.ORCID https://orcid.org/0000-0003-0645-6028
Severance Hospital · KR

Funding

JW Pharmaceutical Corporation 4-2017-0450Korean Fund for Regenerative Medicine 21A0202L1National Research Foundation of Korea NRF2013R1A1A2005083Yonsei University College of Medicine 6-2017-0063
6 · The paper itself

Abstract

backgroundPreeclampsia (PE) is known to arise from insufficient trophoblast invasion as uterine spiral arteries lack remodeling. A significant reduction in placental perfusion induces an ischemic placental microenvironment due to reduced oxygen delivery to the placenta and fetus, leading to oxidative stress. Mitochondria are involved in the regulation of cellular metabolism and the production of reactive oxygen species (ROS). NME/NM23 nuceloside diphosphate kinase 4 (

methodsTranscriptome analysis using TSLCs was performed to identify the candidate gene associated with the possible pathophysiology of PE. Then, the expression of

resultsIn patients with PE,

conclusionOur study showed that the expression of the

Indexed as

Pre-EclampsiaTrophoblastsFemaleHumansInfant, NewbornLeukocytes, MononuclearNucleoside Diphosphate Kinase DPlacentaPolyphosphatesPregnancyReactive Oxygen SpeciesTumor Suppressor Protein p53NME4 protein, humanNucleoside Diphosphate Kinase DPolyphosphatesReactive Oxygen Speciestriphosphoric acidTumor Suppressor Protein p53Cell DeathInduced Pluripotent Stem CellsMitochondrial MatrixPreeclampsiaTranscriptome AnalysisTrophoblast Stem-Like Cells

Identifiers

PMID37096311
PMCPMC10125796
OpenAlexW4362689378

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.