Evidence map›Paper›PMID 37090692›Full record

ReviewFrontiers in immunology2023

New insights into autophagy in inflammatory subtypes of asthma.

Hongna Dong, Wei Yang, Wei Li, Simin Zhu, Ling Zhu, Peng Gao, Yuqiu Hao

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in immunology, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Crosstalk Between Allergic Inflammation and Autophagy.International journal of molecular sciences · 2025
    Review
  6. [Nan fang yi ke da xue xue bao = Journal of Southern Medical University · 2025
    Article
  7. Article
  8. Food science & nutrition · 2025
    Article
  9. Article
  10. Review
  11. Review
  12. Article
  13. Autophagy in chronic rhinosinusitis with or without nasal polyps.Frontiers in cell and developmental biology · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Hongna DongDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Wei YangDepartment of Immunology, College of Basic Medical Sciences, Jilin University, Changchun, China.
Wei LiDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Simin ZhuDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Ling ZhuDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Peng GaoDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Yuqiu HaoDepartment of Respiratory Medicine, The Second Hospital of Jilin University, Changchun, Jilin, China.
Second Affiliated Hospital of Jilin University · CNJilin Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Asthma is a heterogeneous airway disease characterized by airway inflammation and hyperresponsiveness. Autophagy is a self-degrading process that helps maintain cellular homeostasis. Dysregulation of autophagy is involved in the pathogenesis of many diseases. In the context of asthma, autophagy has been shown to be associated with inflammation, airway remodeling, and responsiveness to drug therapy. In-depth characterization of the role of autophagy in asthma can enhance the understanding of the pathogenesis, and provide a theoretical basis for the development of new biomarkers and targeted therapy for asthma. In this article, we focus on the relationship of autophagy and asthma, and discuss its implications for asthma pathogenesis and treatment.

Indexed as

AsthmaAutophagyBiomarkersHumansInflammationRespiratory SystemBiomarkersasthmaautophagybiomarkerinflammationtherapeutic target

Identifiers

PMID37090692
PMCPMC10117973
OpenAlexW4362671633

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.