Evidence map›Paper›PMID 37081068›Full record

ArticleScientific reports2023

Molecular mechanism underlying the increased risk of colorectal cancer metastasis caused by single nucleotide polymorphisms in LI-cadherin gene.

Anna Yui, Daisuke Kuroda, Takahiro Maruno, Makoto Nakakido, Satoru Nagatoishi, Susumu Uchiyama, Kouhei Tsumoto

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Scientific reports, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.6field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 2 citations in OpenAlex.

  1. Cadherin 17 and digestive cancers: from diagnostic to therapeutic opportunities.Journal of experimental & clinical cancer research : CR · 2026
    Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Anna YuiDepartment of Bioengineering, School of Engineering, The University of Tokyo, Tokyo, Japan.
Daisuke KurodaMedical Device Development and Regulation Research Center, School of Engineering, The University of Tokyo, Tokyo, Japan.
Takahiro MarunoDepartment of Biotechnology, Graduate School of Engineering, Osaka University, Osaka, Japan.
Makoto NakakidoDepartment of Bioengineering, School of Engineering, The University of Tokyo, Tokyo, Japan.
Satoru NagatoishiInstitute of Medical Science, The University of Tokyo, Tokyo, Japan.
Susumu UchiyamaDepartment of Biotechnology, Graduate School of Engineering, Osaka University, Osaka, Japan.
Kouhei TsumotoDepartment of Bioengineering, School of Engineering, The University of Tokyo, Tokyo, Japan. tsumoto@bioeng.t.u-tokyo.ac.jp.
The University of Tokyo · JPOsaka University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

LI-cadherin is a member of the cadherin superfamily. LI-cadherin mediates Ca

Indexed as

Colorectal NeoplasmsPolymorphism, Single NucleotideCadherinsCell AdhesionHumansLymphatic MetastasisCadherinsCDH17 protein, human

Identifiers

PMID37081068
PMCPMC10117238
OpenAlexW4366590131

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.