Evidence map›Paper›PMID 37069341›Full record

ArticleScientific reports2023

Phosphatidylinositol metabolism of the renal proximal tubule S3 segment is disturbed in response to diabetes.

Rosalie G J Rietjens, Gangqi Wang, Anouk I M van der Velden, Angela Koudijs, M Cristina Avramut, Sander Kooijman, Patrick C N Rensen, Johan van der Vlag, Ton J Rabelink, Bram Heijs and 1 more

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
2.4field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Rosalie G J RietjensDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands.
Gangqi WangDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands.
Anouk I M van der VeldenDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands.
Angela KoudijsDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands.
M Cristina AvramutEinthoven Laboratory of Vascular and Regenerative Medicine, Leiden University Medical Center, Leiden, The Netherlands.
Sander KooijmanEinthoven Laboratory of Vascular and Regenerative Medicine, Leiden University Medical Center, Leiden, The Netherlands.
Patrick C N RensenEinthoven Laboratory of Vascular and Regenerative Medicine, Leiden University Medical Center, Leiden, The Netherlands.
Johan van der VlagDepartment of Nephrology, Radboud University Medical Center, Nijmegen, The Netherlands.
Ton J RabelinkDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands.
Bram HeijsThe Novo Nordisk Foundation Center for Stem Cell Medicine (reNEW), Leiden University Medical Center, Leiden, The Netherlands.
Bernard M van den BergDepartment of Internal Medicine (Nephrology), Leiden University Medical Center, Leiden, The Netherlands. b.m.van_den_berg@lumc.nl.
Leiden University Medical Center · NLNetherlands Metabolomics Centre · NLRadboud University Medical Center · NL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diabetes is a main risk factor for kidney disease, causing diabetic nephropathy in close to half of all patients with diabetes. Metabolism has recently been identified to be decisive in cell fate decisions and repair. Here we used mass spectrometry imaging (MSI) to identify tissue specific metabolic dysregulation, in order to better understand early diabetes-induced metabolic changes of renal cell types. In our experimental diabetes mouse model, early glomerular glycocalyx barrier loss and systemic metabolic changes were observed. In addition, MSI targeted at small molecule metabolites and glycero(phospho)lipids exposed distinct changes upon diabetes in downstream nephron segments. Interestingly, the outer stripe of the outer medullar proximal tubular segment (PT_S3) demonstrated the most distinct response compared to other segments. Furthermore, phosphatidylinositol lipid metabolism was altered specifically in PT_S3, with one of the phosphatidylinositol fatty acid tails being exchanged from longer unsaturated fatty acids to shorter, more saturated fatty acids. In acute kidney injury, the PT_S3 segment and its metabolism are already recognized as important factors in kidney repair processes. The current study exposes early diabetes-induced changes in membrane lipid composition in this PT_S3 segment as a hitherto unrecognized culprit in the early renal response to diabetes.

Indexed as

Diabetes MellitusDiabetic NephropathiesAnimalsKidneyKidney TubulesKidney Tubules, ProximalLipid MetabolismMice

Identifiers

PMID37069341
PMCPMC10110589
OpenAlexW4366158534

What OpenQuestion holds

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LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.