Evidence map›Paper›PMID 37048809›Full record

ReviewJournal of clinical medicine2023

Secondary IgA Nephropathy and IgA-Associated Nephropathy: A Systematic Review of Case Reports.

Maciej Tota, Vanessa Baron, Katie Musial, Bouchra Derrough, Andrzej Konieczny, Magdalena Krajewska, Kultigin Turkmen, Mariusz Kusztal

Open access · goldAbstract readReview
In one paragraph

Review in Journal of clinical medicine, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
3.4field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 17 citations in OpenAlex.

  1. Article
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  12. Acute kidney injury after treatment with arsenic trioxide.Clinical nephrology. Case studies · 2025
    Article
  13. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 2 countries.

Maciej TotaFaculty of Medicine, Wroclaw Medical University, 50-367 Wrocław, Poland.ORCID 0000-0002-5298-6479
Vanessa BaronFaculty of Medicine, Wroclaw Medical University, 50-367 Wrocław, Poland.ORCID 0000-0003-4602-3024
Katie MusialFaculty of Medicine, Wroclaw Medical University, 50-367 Wrocław, Poland.
Bouchra DerroughFaculty of Medicine, Wroclaw Medical University, 50-367 Wrocław, Poland.ORCID 0000-0003-1545-9511
Andrzej KoniecznyDepartment of Nephrology and Transplantation Medicine, Wroclaw Medical University, 50-556 Wrocław, Poland.ORCID 0000-0002-4966-9771
Magdalena KrajewskaDepartment of Nephrology and Transplantation Medicine, Wroclaw Medical University, 50-556 Wrocław, Poland.ORCID 0000-0002-2632-2409
Kultigin TurkmenDivision of Nephrology, Department of Internal Medicine, Meram Medical Faculty, Necmettin Erbakan University, Konya 42090, Turkey.ORCID 0000-0002-1667-7716
Mariusz KusztalDepartment of Nephrology and Transplantation Medicine, Wroclaw Medical University, 50-556 Wrocław, Poland.ORCID 0000-0002-6502-0374
Wroclaw Medical University · PLNecmettin Erbakan University · TR

Funding

Wroclaw Medical University SUBZ.C160.22.051
6 · The paper itself

Abstract

Primary (pIgAN), secondary IgA nephropathy (sIgAN), and IgA-associated nephropathy can be distinguished. While pIgAN has been thoroughly studied, information about the etiology of sIgAN remains scarce. As concerns sIgAN, several studies suggest that different etiologic factors play a role and ultimately lead to a pathophysiologic process similar to that of pIgAN. In this article, we review a vast number of cases in order to determine the novel putative underlying diseases of sIgAN. Moreover, updates on the common pathophysiology of primary disorders and sIgAN are presented. We identified liver, gastrointestinal, oncological, dermatological, autoimmune, and respiratory diseases, as well as infectious, iatrogenic, and environmental factors, as triggers of sIgAN. As novel biological therapies for listed underlying diseases emerge, we suggest implementing drug-induced sIgAN as a new significant category. Clinicians should acknowledge the possibility of sIgAN progression in patients treated with TNF-α inhibitors, IL-12/IL-23-inhibitors, immune checkpoint inhibitors, CTLA-4, oral anticoagulants, thioureylene derivatives, and anti-vascular endothelial growth factor drugs.

Indexed as

autoimmunecancerdermatologicaldrug-inducedgastrointestinalIgA-associatedinfectionliverpathophysiologytreatment

Identifiers

PMID37048809
PMCPMC10094848
OpenAlexW4362670626

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.