Evidence map›Paper›PMID 37003030›Full record

ArticleLeukemia research2023

Rituximab induced cytokine release with high serum IP-10 (CXCL10) concentrations is associated with infusion reactions.

Jeremiah E Moore, Paige C Bloom, Charles C Chu, Jennifer E Bruno, Christine A Herne, Andrea M Baran, Sally A Quataert, Timothy R Mosmann, Ronald P Taylor, Danielle S Wallace and 3 more

Open access · greenAbstract read
In one paragraph

Article in Leukemia research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
1.7field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 6 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 2 institutions in 1 country.

Jeremiah E MooreJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States; Department of Pharmacy, University of Rochester Medical Center, Rochester, NY, United States.
Paige C BloomJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States.
Charles C ChuJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States; Division of Hematology/Oncology, Department of Medicine, University of Rochester Medical Center, Rochester, NY, United States.
Jennifer E BrunoCenter for Vaccine Biology & Immunology, University of Rochester Medical Center, Rochester, NY, United States; Department of Microbiology and Immunology, University of Rochester Medical Center, Rochester, NY, United States.
Christine A HerneJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States.
Andrea M BaranDepartment of Biostatistics and Computational Biology, University of Rochester Medical Center, Rochester, NY, United States.
Sally A QuataertCenter for Vaccine Biology & Immunology, University of Rochester Medical Center, Rochester, NY, United States; Department of Microbiology and Immunology, University of Rochester Medical Center, Rochester, NY, United States.
Timothy R MosmannCenter for Vaccine Biology & Immunology, University of Rochester Medical Center, Rochester, NY, United States; Department of Microbiology and Immunology, University of Rochester Medical Center, Rochester, NY, United States.
Ronald P TaylorDepartment of Biochemistry and Molecular Genetics, University of Virginia School of Medicine, Charlottesville, VA, United States.
Danielle S WallaceJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States; Division of Hematology/Oncology, Department of Medicine, University of Rochester Medical Center, Rochester, NY, United States.
Michael R ElliottCenter for Cell Clearance and the Department of Microbiology, Immunology, and Cancer Biology, University of Virginia, Charlottesville, VA, United States.
Paul M BarrJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States; Division of Hematology/Oncology, Department of Medicine, University of Rochester Medical Center, Rochester, NY, United States.
Clive S ZentJames P. Wilmot Cancer Institute, University of Rochester Medical Center, Rochester, NY, United States; Division of Hematology/Oncology, Department of Medicine, University of Rochester Medical Center, Rochester, NY, United States. Electronic address: clive_zent@urmc.rochester.edu.
University of Rochester Medical Center · USUniversity of Virginia · US

Funding

Re-designing anti-CD20 monoclonal antibody therapy using novel insights into limitations of innate immune cytotoxicityR21CA267040 · NCI · UNIVERSITY OF ROCHESTER · PI ZENT, CLIVE STEVEN · 2022 to 2023
$392k
NCI NIH HHS R21 CA267040
6 · The paper itself

Abstract

Monoclonal antibody induced infusion reactions (IRs) can be serious and even fatal. We used clinical data and blood samples from 37 treatment naïve patients with chronic lymphocytic leukemia/small lymphocytic lymphoma (CLL) initiating therapy for progressive disease with a single 50 mg dose of intravenous (IV) rituximab at 25 mg/h. Twenty-four (65 %) patients had IRs at a median of 78 min (range 35-128) and rituximab dose of 32 mg (range 15-50). IR risk did not correlate with patient or CLL characteristics, CLL counts or CD20 levels, or serum rituximab or complement concentrations. Thirty-five (95 %) patients had cytokine release response with a ≥ 4-fold increase in serum concentration of ≥ 1 inflammatory cytokine. IRs were associated with significantly higher post-infusion serum concentrations of gamma interferon induced cytokines IP-10, IL-6 and IL-8. IP-10 concentrations increased ≥ 4-fold in all patients with an IR and were above the upper limit of detection (40,000 pg/ml) in 17 (71 %). In contrast, to only three (23 %) patients without an IR had an ≥ 4-fold increase in serum concentrations of IP-10 (highest 22,013 pg/ml). Our data suggest that cytokine release could be initiated by activation of effector cells responsible for clearance of circulating CLL cells with IRs occurring in those with higher levels of gamma interferon induced cytokines. These novel insights could inform future research to better understand and manage IRs and understand the role of cytokines in the control of cytotoxic immune responses to mAb.

Indexed as

Antineoplastic AgentsLeukemia, Lymphocytic, Chronic, B-CellAntibodies, Monoclonal, Murine-DerivedChemokine CXCL10CytokinesHumansInterferon-gammaRituximabAntibodies, Monoclonal, Murine-DerivedAntineoplastic AgentsChemokine CXCL10CXCL10 protein, humanCytokinesInterferon-gammaRituximabChronic lymphocytic leukemiaCXCL10Cytokine release syndromeFirst dose infusion reactionGamma interferonIP-10Rituximab

Identifiers

PMID37003030
PMCPMC10219853
OpenAlexW4361284177

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.