Evidence map›Paper›PMID 36986224›Full record

ReviewNutrients2023

Interactions between Intestinal Homeostasis and NAD

Taichi Nagahisa, Shotaro Kosugi, Shintaro Yamaguchi

Open access · goldAbstract readReview
In one paragraph

Review in Nutrients, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.0field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 11 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Review
  5. Article
  6. Review
  7. Review
  8. Article
  9. The Gut Microbiota Affects Anti-TNF Responsiveness by Activating the NADAdvanced science (Weinheim, Baden-Wurttemberg, Germany) · 2025
    Article
  10. Article
  11. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Taichi NagahisaDivision of Endocrinology, Metabolism and Nephrology, Department of Internal Medicine, Keio University School of Medicine, Shinjuku-ku, Tokyo 160-8582, Japan.
Shotaro KosugiDivision of Endocrinology, Metabolism and Nephrology, Department of Internal Medicine, Keio University School of Medicine, Shinjuku-ku, Tokyo 160-8582, Japan.
Shintaro YamaguchiDivision of Endocrinology, Metabolism and Nephrology, Department of Internal Medicine, Keio University School of Medicine, Shinjuku-ku, Tokyo 160-8582, Japan.ORCID 0000-0002-4917-033X
Keio University · JP

Funding

Daiwa Securities Health Foundation 2022Inamori Research Grant Program 2021Keio University Global Research Institute Pre-Start-up Grant 2022Scientific Research Fund of the Ministry of Education, Culture, Sports, Science, and Technology of Japan 21K07377Takeda Science Foundation 2019
6 · The paper itself

Abstract

The intestine has garnered attention as a target organ for developing new therapies for impaired glucose tolerance. The intestine, which produces incretin hormones, is the central regulator of glucose metabolism. Glucagon-like peptide-1 (GLP-1) production, which determines postprandial glucose levels, is regulated by intestinal homeostasis. Nicotinamide phosphoribosyltransferase (NAMPT)-mediated nicotinamide adenine dinucleotide (NAD

Indexed as

Glucose IntoleranceNADAMP-Activated Protein KinasesBiologyCytokinesGlucagon-Like Peptide 1GlucoseHomeostasisHumansIncretinsNicotinamide PhosphoribosyltransferaseObesityAMP-Activated Protein KinasesCytokinesGlucagon-Like Peptide 1GlucoseIncretinsNADNicotinamide Phosphoribosyltransferaseincretinintestinal homeostasisnicotinamide adenine dinucleotidenicotinamide phosphoribosyltransferasepostprandial glucose metabolism

Identifiers

PMID36986224
PMCPMC10052115
OpenAlexW4328119912

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.