Evidence map›Paper›PMID 36979813›Full record

ReviewBiomedicines2023

Dilated Cardiomyopathy: A Comprehensive Approach to Diagnosis and Risk Stratification.

André Ferreira, Vera Ferreira, Miguel Marques Antunes, Ana Lousinha, Tiago Pereira-da-Silva, Diana Antunes, Pedro Silva Cunha, Mário Oliveira, Rui Cruz Ferreira, Sílvia Aguiar Rosa

Open access · goldFull text readReview
In one paragraph

Review in Biomedicines, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
6.6field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 30 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Review
  6. Review
  7. Article
  8. Review
  9. Review
  10. Review
  11. Review
  12. Article
  13. Article
  14. Article
  15. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

André FerreiraCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.ORCID 0000-0002-8762-9551
Vera FerreiraCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.ORCID 0000-0002-8957-9279
Miguel Marques AntunesCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.ORCID 0000-0002-0887-8219
Ana LousinhaCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.
Tiago Pereira-da-SilvaCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.ORCID 0000-0003-0467-2608
Diana AntunesDepartment of Genetics, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.
Pedro Silva CunhaCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.ORCID 0000-0001-9465-634X
Mário OliveiraCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.
Rui Cruz FerreiraCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.
Sílvia Aguiar RosaCardiology Service, Hospital Santa Marta, Centro Hospitalar Universitário de Lisboa Central, 1169-050 Lisbon, Portugal.
Hospital de Santa Marta · PTUnidade Local de Saúde de São José · PT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Dilated cardiomyopathy (DCM) represents one of the most common causes of non-ischemic heart failure, characterised by ventricular dilation alongside systolic dysfunction. Despite advances in therapy, DCM mortality rates remain high, and it is one of the leading causes of heart transplantation. It was recently recognised that many patients present minor structural cardiac abnormalities and express different arrhythmogenic phenotypes before overt heart-failure symptoms. This has raised several diagnostic and management challenges, including the differential diagnosis with other phenotypically similar conditions, the identification of patients at increased risk of malignant arrhythmias, and of those who will have a worse response to medical therapy. Recent developments in complementary diagnostic procedures, namely cardiac magnetic resonance and genetic testing, have shed new light on DCM understanding and management. The present review proposes a comprehensive and systematic approach to evaluating DCM, focusing on an improved diagnostic pathway and a structured stratification of arrhythmic risk that incorporates novel imaging modalities and genetic test results, which are critical for guiding clinical decision-making and improving outcomes.

Indexed as

arrhythmogenic left ventricle cardiomyopathycardiovascular magnetic resonancedilated cardiomyopathygenetic testingreverse remodellingrisk stratification

Identifiers

PMID36979813
PMCPMC10044994
OpenAlexW4323841791

What OpenQuestion holds

Textfull text, public
LicenceCC BY
measurements read30
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.