Evidence map›Paper›PMID 36979413›Full record

ReviewBiomolecules2023

Antonis Roussos, Katerina Kitopoulou, Fivos Borbolis, Konstantinos Palikaras

Open access · goldFull text readReview
In one paragraph

Review in Biomolecules, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 42 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
42citing papers in PubMed, 1 pooled it
5.5field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

42 citing papers in PubMed, 1 synthesis or guideline pooled it, 62 citations in OpenAlex.

  1. Pooled it
  2. International journal of molecular sciences · 2026
    Review
  3. Article
  4. Review
  5. [Bushen Yijing Formula attenuates amyloid β-protein deposi-tion and oxidative damage inZhejiang da xue xue bao. Yi xue ban = Journal of Zhejiang University. Medical sciences · 2026
    Article
  6. Article
  7. Usefulness ofInternational journal of molecular sciences · 2026
    Article
  8. bioRxiv : the preprint server for biology · 2026
    Article
  9. Article
  10. High-Throughput Toxicity Screening withEnvironmental science & technology · 2026
    Review
  11. Article
  12. Frontiers in aging neuroscience · 2026
    Review
  13. Article
  14. Dietary mycotoxin toxicity inAdvances in neurotoxicology · 2026
    Article
  15. Review
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Antonis RoussosDepartment of Physiology, Medical School, National and Kapodistrian University of Athens, GR-157 27 Athens, Greece.
Katerina KitopoulouDepartment of Physiology, Medical School, National and Kapodistrian University of Athens, GR-157 27 Athens, Greece.
Fivos BorbolisDepartment of Physiology, Medical School, National and Kapodistrian University of Athens, GR-157 27 Athens, Greece.ORCID 0000-0001-6559-1356
Konstantinos PalikarasDepartment of Physiology, Medical School, National and Kapodistrian University of Athens, GR-157 27 Athens, Greece.ORCID 0000-0001-6992-5560
National and Kapodistrian University of Athens · GRUniversity of Padua · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In recent years, advances in science and technology have improved our quality of life, enabling us to tackle diseases and increase human life expectancy. However, longevity is accompanied by an accretion in the frequency of age-related neurodegenerative diseases, creating a growing burden, with pervasive social impact for human societies. The cost of managing such chronic disorders and the lack of effective treatments highlight the need to decipher their molecular and genetic underpinnings, in order to discover new therapeutic targets. In this effort, the nematode

Indexed as

Caenorhabditis elegansNeurodegenerative DiseasesAnimalsDisease Models, AnimalHumansQuality of LifeageingAlzheimer’s diseaseamyotrophic lateral sclerosisAutosomal Dominant Optic AtrophyC. elegansCockayne syndromeHuntington’s diseasemitochondrianeurodegenerationParkinson’s disease

Identifiers

PMID36979413
PMCPMC10046667
OpenAlexW4323313435

What OpenQuestion holds

Textfull text, public
LicenceCC BY
measurements read12
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.