Evidence map›Paper›PMID 36904234›Full record

ReviewNutrients2023

Focusing on Phosphorus Loads: From Healthy People to Chronic Kidney Disease.

Mengjing Wang, Jiaying Zhang, Kamyar Kalantar-Zadeh, Jing Chen

Full text readReview
In one paragraph

Review in Nutrients, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed.

  1. Article
  2. Planetary Health Diet: Emphasizing Nutritional Therapy in Kidney Disease.Clinical journal of the American Society of Nephrology : CJASN · 2026
    Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Review
  12. Article
  13. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Mengjing WangNephrology, Huashan Hospital, Fudan University, Shanghai 200040, China.ORCID 0000-0003-4201-4534
Jiaying ZhangNutritional Department, Huashan Hospital, Fudan University, Shanghai 200040, China.
Kamyar Kalantar-ZadehHarold Simmons Center for Kidney Disease Research and Epidemiology, Division of Nephrology and Hypertension, University of California Irvine Medical Center, Orange, CA 92868, USA.ORCID 0000-0002-8666-0725
Jing ChenNephrology, Huashan Hospital, Fudan University, Shanghai 200040, China.

Funding

China natural science grant 81730017Clinical Technology Innovation Project of Shanghai Shenkang Hospital Development Center SHDC12018111Key research and development plan of the Ministry of Science and Technology 2020YFC2005003Shenkang three-year action plan project SHDC2020CR4014
6 · The paper itself

Abstract

Phosphorus is an essential micromineral with a key role in cellular metabolism and tissue structure. Serum phosphorus is maintained in a homeostatic range by the intestines, bones, and kidneys. This process is coordinated by the endocrine system through the highly integrated actions of several hormones, including FGF23, PTH, Klotho, and 1,25D. The excretion kinetics of the kidney after diet phosphorus load or the serum phosphorus kinetics during hemodialysis support that there is a "pool" for temporary phosphorus storage, leading to the maintenance of stable serum phosphorus levels. Phosphorus overload refers to a state where the phosphorus load is higher than is physiologically necessary. It can be caused by a persistently high-phosphorus diet, renal function decline, bone disease, insufficient dialysis, and inappropriate medications, and includes but is not limited to hyperphosphatemia. Serum phosphorus is still the most commonly used indicator of phosphorus overload. Trending phosphorus levels to see if they are chronically elevated is recommended instead of a single test when judging phosphorus overload. Future studies are needed to validate the prognostic role of a new marker or markers of phosphorus overload.

Indexed as

Phosphorus, DietaryRenal Insufficiency, ChronicBone and BonesFibroblast Growth FactorsHumansKidneyPhosphorusFibroblast Growth FactorsPhosphorusPhosphorus, Dietarychronic kidney diseasephosphorus loadphosphorus pool

Identifiers

PMID36904234
PMCPMC10004810

What OpenQuestion holds

Textfull text, public
LicenceCC BY
measurements read25
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.