Evidence map›Paper›PMID 36901899›Full record

ReviewInternational journal of molecular sciences2023

Alicja Rabiasz, Ewa Ziętkiewicz

Full text readReview
In one paragraph

Review in International journal of molecular sciences, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. Knockdown ofmicroPublication biology · 2026
    Article
  3. Article
  4. Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Alicja RabiaszInstitute of Human Genetics Polish Academy of Sciences, 32 Strzeszyńska Street, 60-479 Poznań, Poland.ORCID 0000-0003-3365-4812
Ewa ZiętkiewiczInstitute of Human Genetics Polish Academy of Sciences, 32 Strzeszyńska Street, 60-479 Poznań, Poland.ORCID 0000-0003-2437-5088

Funding

National Science Center 2018/29/N/NZ5/00810 (A.R.)National Science Center 2018/31/B/NZ2/03248 (E.Z.)
6 · The paper itself

Abstract

Cilia and flagella are evolutionarily conserved organelles that form protrusions on the surface of many growth-arrested or differentiated eukaryotic cells. Due to the structural and functional differences, cilia can be roughly classified as motile and non-motile (primary). Genetically determined dysfunction of motile cilia is the basis of primary ciliary dyskinesia (PCD), a heterogeneous ciliopathy affecting respiratory airways, fertility, and laterality. In the face of the still incomplete knowledge of PCD genetics and phenotype-genotype relations in PCD and the spectrum of PCD-like diseases, a continuous search for new causative genes is required. The use of model organisms has been a great part of the advances in understanding molecular mechanisms and the genetic basis of human diseases; the PCD spectrum is not different in this respect. The planarian model (

Indexed as

CiliopathiesPlanariansAnimalsCiliaFlagellaHumansMediterraneaMutationcandidate genesflatwormsmotile ciliaplanariansprimary ciliary dyskinesia (PCD)RNA interference (RNAi)

Identifiers

PMID36901899
PMCPMC10002865

What OpenQuestion holds

Textfull text, public
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measurements read3
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.