Evidence map›Paper›PMID 36831297›Full record

ArticleCells2023

Evidence for Epistatic Interaction between

Maria Luiza de Oliveira-Caramez, Luciana Veiga-Castelli, Andreia S Souza, Renata Nahas Cardili, David Courtin, Milena Flória-Santos, Eduardo Donadi, Silvana Giuliatti, Audrey Sabbagh, Erick C Castelli and 1 more

Open access · goldAbstract read
In one paragraph

Article in Cells, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed, 1 pooled it
1.2field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 1 synthesis or guideline pooled it, 7 citations in OpenAlex.

  1. Evaluation ofMedicina (Kaunas, Lithuania) · 2023
    Pooled it
  2. Article
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 2 countries.

Maria Luiza de Oliveira-CaramezDepartamento de Genética, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.ORCID 0000-0002-8155-0244
Luciana Veiga-CastelliDepartamento de Genética, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.
Andreia S SouzaMolecular Genetics and Bioinformatics Laboratory, School of Medicine, São Paulo State University (UNESP), Botucatu 18618-687, SP, Brazil.ORCID 0000-0002-6853-1559
Renata Nahas CardiliDepartamento de Clínica Médica, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.
David CourtinInstitut de Recherche pour le Développement, UMR 261 MERIT, Université de Paris, F-75006 Paris, France.
Milena Flória-SantosDepartamento de Enfermagem Materno-Infantil e Saúde Pública-(EERP/ERM), Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.ORCID 0000-0002-0656-1736
Eduardo DonadiDepartamento de Clínica Médica, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.
Silvana GiuliattiDepartamento de Genética, Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, Brazil.ORCID 0000-0002-7520-6420
Audrey SabbaghInstitut de Recherche pour le Développement, UMR 261 MERIT, Université de Paris, F-75006 Paris, France.
Erick C CastelliMolecular Genetics and Bioinformatics Laboratory, School of Medicine, São Paulo State University (UNESP), Botucatu 18618-687, SP, Brazil.
Celso Teixeira Mendes-JuniorLaboratório de Pesquisas Forenses e Genômicas, Departamento de Química, Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14040-901, SP, Brazil.ORCID 0000-0002-7337-1203
Universidade de Ribeirão Preto · BRUniversidade Estadual Paulista (Unesp) · BRUniversité Paris Cité · FRUniversidade de São Paulo · BR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Vitiligo is the most frequent cause of depigmentation worldwide. Genetic association studies have discovered about 50 loci associated with disease, many with immunological functions. Among them is HLA-G, which modulates immunity by interacting with specific inhibitory receptors, mainly LILRB1 and LILRB2. Here we investigated the

Indexed as

Antigens, CDHLA-G AntigensLeukocyte Immunoglobulin-like Receptor B1VitiligoHumansPolymorphism, GeneticReceptors, ImmunologicAntigens, CDHLA-G AntigensLeukocyte Immunoglobulin-like Receptor B1LILRB1 protein, humanReceptors, ImmunologicancestryBrazilILT-2ILT-4LILRB2SNPforID

Identifiers

PMID36831297
PMCPMC9954564
OpenAlexW4321096361

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.