Evidence map›Paper›PMID 36750303›Full record

ArticleBMJ case reports2023

Total colectomy for poorly controlled hypokalaemia due to Gitelman syndrome.

Tetsuya Kawahara, Tetsuya Inazu, Shingo Ishida

Open access · greenAbstract readCase Reports
In one paragraph

Article in BMJ case reports, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 99% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Tetsuya KawaharaEndocrinology and Metabolism, Shin Komonji Hospital, Kitakyushu, Fukuoka, Japan k-tetsuy@med.uoeh-u.ac.jp.ORCID http://orcid.org/0000-0002-1268-4871
Tetsuya InazuCollege of Pharmaceutical Science, Ritsumeikan University-Biwako Kusatsu Campus, Kusatsu, Shiga, Japan.
Shingo IshidaSurgery, Shin Komonji Hospital, Kitakyushu, Fukuoka, Japan.
Uji Hospital · JPRitsumeikan University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Gitelman syndrome (GS) is an autosomal recessive tubulopathy caused by dysfunction of the thiazide-sensitive sodium-chloride cotransporter, which leads to hypokalaemia, metabolic alkalosis, hypomagnesaemia and hypocalciuria. Patients with GS show varied clinical features due to hypokalaemia: tetany, muscle weakness, periodical paralysis and constipation, which is one of the most frequent ones. This paper presents the case of a woman in her 40s referred to our endocrinology department for severe hypokalaemia. After biochemical and genetic analyses, a diagnosis of GS was established. Concurrently, the patient suffered from refractory constipation due to hypokalaemia and underwent a total colectomy with ileorectal anastomosis, which cured both disorders without any medication for 3 years.

Indexed as

AlkalosisGitelman SyndromeHypokalemiaColectomyFemaleHumansSolute Carrier Family 12, Member 3Solute Carrier Family 12, Member 3EndocrinologyFluid electrolyte and acid-base disturbancesGastroenterology

Identifiers

PMID36750303
PMCPMC9906164
OpenAlexW4319344919

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.