Evidence map›Paper›PMID 36731630›Full record

ReviewVirus research2023

Role of genetic factors in different swine breeds exhibiting varying levels of resistance/susceptibility to PRRSV.

Xiangbin You, Gan Li, Ying Lei, Zhiqian Xu, Ping Zhang, Youbing Yang

Open access · goldAbstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Virus research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
2.8field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 15 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Host factorFrontiers in immunology · 2024
    Article
  9. Article
  10. Porcine Reproductive and Respiratory Syndrome (PRRS) andAnimals : an open access journal from MDPI · 2023
    Article
  11. Review
  12. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Xiangbin YouCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China.
Gan LiCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China.
Ying LeiCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China.
Zhiqian XuCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China.
Ping ZhangCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China.
Youbing YangCollege of Animal Science and Technology, Henan University of Science and Technology, Luoyang 471023, China; Luoyang Key Laboratory of Animal Genetics and Breeding, Luoyang 471023, China. Electronic address: 9906133@haust.edu.cn.
Henan University of Science and Technology · CNLuoyang Institute of Science and Technology · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Porcine reproductive and respiratory syndrome (PRRS), caused by the PRRS virus (PRRSV), is an economically significant contagious disease. Traditional approaches based on vaccines or medicines were challenging to control PRRSV due to the diversity of viruses. Different breeds of pigs infected with PRRSV have been reported to have different immune responses. However, due to the complexity of interaction mechanism between host and PRRSV, the genetic mechanism leading to PRRSV susceptibility/resistance in various pig breeds is still unclear. Herein, the role of host genetic components in PRRSV susceptibility is systematically described, and the molecular mechanisms by which host genetic factors such as SNPs, cytokines, receptor molecules, intestinal flora, and non-coding RNAs regulate PRRSV susceptibility/resistance. Therefore, improving the resistance to disease of individual animals through disease-resistance breeding technology is of profound significance for uplifting the sustainable and healthy development of the pig industry.

Indexed as

Porcine Reproductive and Respiratory SyndromePorcine respiratory and reproductive syndrome virusAnimalsCytokinesSwineCytokinesGut microbiotaNon-coding RNAsPigPRRSVReceptorResistance/susceptibilitySNPs

Identifiers

PMID36731630
PMCPMC10194364
OpenAlexW4319057605

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.