Evidence map›Paper›PMID 36699859›Full record

ArticleFrontiers in plant science2022

Combined metabolomic and transcriptomic analysis reveals key components of

Shuai Lu, Yaoyu Chen, Surong Wang, Binying Han, Chenglei Zhao, Penghui Xue, Yue Zhang, Hui Fang, Baohua Wang, Yunying Cao

Open access · goldAbstract read
In one paragraph

Article in Frontiers in plant science, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
17.9field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 25 citations in OpenAlex.

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  5. Comparative genomics and expression analysis of theFrontiers in plant science · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 1 institution in 1 country.

Shuai LuSchool of Life Sciences, Nantong University, Nantong, China.
Yaoyu ChenSchool of Life Sciences, Nantong University, Nantong, China.
Surong WangSchool of Life Sciences, Nantong University, Nantong, China.
Binying HanSchool of Life Sciences, Nantong University, Nantong, China.
Chenglei ZhaoSchool of Life Sciences, Nantong University, Nantong, China.
Penghui XueSchool of Life Sciences, Nantong University, Nantong, China.
Yue ZhangSchool of Life Sciences, Nantong University, Nantong, China.
Hui FangSchool of Life Sciences, Nantong University, Nantong, China.
Baohua WangSchool of Life Sciences, Nantong University, Nantong, China.
Yunying CaoSchool of Life Sciences, Nantong University, Nantong, China.
Nantong University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Oryza Sativa is one of the most important food crops in China, which is easily affected by drought during its growth and development. As a member of the calcium signaling pathway, CBL-interacting protein kinase (CIPK) plays an important role in plant growth and development as well as environmental stress. However, there is no report on the function and mechanism of OsCIPK17 in rice drought resistance. We combined transcriptional and metabonomic analysis to clarify the specific mechanism of OsCIPK17 in response to rice drought tolerance. The results showed that OsCIPK17 improved drought resistance of rice by regulating deep roots under drought stress; Response to drought by regulating the energy metabolism pathway and controlling the accumulation of citric acid in the tricarboxylic acid (TCA) cycle; Our exogenous experiments also proved that OsCIPK17 responds to citric acid, and this process involves the auxin metabolism pathway; Exogenous citric acid can improve the drought resistance of overexpression plants. Our research reveals that OsCIPK17 positively regulates rice drought resistance and participates in the accumulation of citric acid in the TCA cycle, providing new insights for rice drought resistance.

Indexed as

auxinCIPKcitric aciddroughtmetabolomeOryza sativatranscriptome

Identifiers

PMID36699859
PMCPMC9868928
OpenAlexW4313909627

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.