Evidence map›Paper›PMID 36626452›Full record

ReviewMedicine2022

Pathomechanisms of SARS-CoV-2 infection and development of atherosclerosis in patients with COVID-19: A review.

Alicja Zofia Gospodarczyk, Celina Wojciechowska, Kamil Piotr Marczewski, Natalia Justyna Gospodarczyk, Jolanta Zalejska-Fiolka

Open access · goldAbstract readReview
In one paragraph

Review in Medicine, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.0field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Alicja Zofia GospodarczykDepartment of Biochemistry, Faculty of Medical Sciences in Zabrze, Medical University of Silesia, Zabrze, Poland.ORCID 0000-0002-2881-508
Celina WojciechowskaSecond Department of Cardiology, Faculty of Medical Sciences in Zabrze, Medical University of Silesia, Zabrze, Poland.
Kamil Piotr MarczewskiDepartment of Emergency Medicine, Faculty of Medical Sciences in Katowice, Medical University of Silesia, Katowice, Poland.
Natalia Justyna GospodarczykDepartment of Emergency Medicine, Faculty of Medical Sciences in Katowice, Medical University of Silesia, Katowice, Poland.
Jolanta Zalejska-FiolkaDepartment of Biochemistry, Faculty of Medical Sciences in Zabrze, Medical University of Silesia, Zabrze, Poland.
Medical University of Silesia · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The pandemic of coronavirus disease 2019 (COVID-19) has posed a major health challenge for over 2 years. The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) that causes it belongs to single-stranded ribonucleic acid viruses and causes acute respiratory distress syndrome. The initial outbreak was discovered in December 2019 in Wuhan province, where SARS-CoV-2 quickly spread to other countries. In addition to respiratory disorders, it has been shown that during and after COVID-19 infection, cardiovascular diseases are often developed or exacerbated, such as: arterial hypertension, coronary artery disease, arrhythmias, heart failure and thromboembolic complications. In view of the higher prevalence of atherosclerosis in patients with COVID-19, we described the pathomechanisms of the development of this infection and the possible correlations between SARS-CoV-2 infection and thromboembolic complications. We focused on the role of the inflammatory response, renin-angiotensin system and endothelial dysfunction in the development of atherosclerosis in patients with COVID-19. A thorough understanding of the hemodynamic mechanisms and the impact of the infection on the cardiovascular system will allow for the proper selection of appropriate therapy in patients after SARS-CoV-2 infection.

Indexed as

AtherosclerosisCardiovascular DiseasesCOVID-19HumansRenin-Angiotensin SystemSARS-CoV-2

Identifiers

PMID36626452
PMCPMC9750607
OpenAlexW4315436583

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.