Evidence map›Paper›PMID 36605901›Full record

ReviewFrontiers in physiology2022

Mitochondrial dysfunction in vascular endothelial cells and its role in atherosclerosis.

Kai Qu, Fang Yan, Xian Qin, Kun Zhang, Wen He, Mingqing Dong, Guicheng Wu

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in physiology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 77 papers.

0numbers the graph read from it
0cells of the map it votes in
77citing papers in PubMed
7.5field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

77 citing papers in PubMed, 98 citations in OpenAlex.

  1. Review
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  4. Observational
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  14. Peripheral blood mononuclear cell mitochondrial bioenergetics are related to vascular endothelial function in young and older adults.American journal of physiology. Regulatory, integrative and comparative physiology · 2026
    Article
  15. Review
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  18. Archives of medical science : AMS · 2026
    Article
  19. Article
  20. Article

17 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Kai QuClinical Research Center for Endocrinology and Metabolic Diseases, Chongqing University Three Gorges Hospital, Chongqing, China.
Fang YanDepartment of Geriatrics, Geriatric Diseases Institute of Chengdu, Chengdu Fifth People's Hospital, Chengdu, Sichuan, China.
Xian QinClinical Research Center for Endocrinology and Metabolic Diseases, Chongqing University Three Gorges Hospital, Chongqing, China.
Kun ZhangClinical Research Center for Endocrinology and Metabolic Diseases, Chongqing University Three Gorges Hospital, Chongqing, China.
Wen HeDepartment of Geriatrics, Clinical trial center, Chengdu Fifth People's Hospital, Chengdu, Sichuan, China.
Mingqing DongCenter for Medicine Research and Translation, Chengdu Fifth People's Hospital, Chengdu, Sichuan, China.
Guicheng WuClinical Research Center for Endocrinology and Metabolic Diseases, Chongqing University Three Gorges Hospital, Chongqing, China.
Chongqing University · CNChengdu Fifth People's Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The mitochondria are essential organelles that generate large amounts of ATP

Indexed as

atherosclerosisendothelial cellsmitochondrial dysfunctionmitochondrial ROSmitophagy

Identifiers

PMID36605901
PMCPMC9807884
OpenAlexW4313432435

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.