Evidence map›Paper›PMID 36560844›Full record

ArticleJournal of genetics2022

Novel combination of

Vivek Kumar, Pramod Kumar, Lakshita Chauhan, Aradhana Dwivedi, H Ravi Ramamurthy

Abstract readCase Reports
PubMed
In one paragraph

Article in Journal of genetics, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact, top 70% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 0 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Vivek KumarDepartment of Pediatrics, Command Hospital, Air Force, Bengaluru 560 007, India. aradhanakd@gmail.com.
Pramod Kumar
Lakshita Chauhan
Aradhana Dwivedi
H Ravi Ramamurthy
Command Hospital Air Force · IN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Pediatric restrictive cardiomyopathy (RCM) is the rarest in its group and accounts for only 2.5-5% of all the diagnosed cardiomyopathies in children. It is a relentless disease with poor prognosis, and heart transplantation is the only long-term treatment option. The aetiology of pediatric RCM varies and includes conditions such as endomyocardial fibrosis, storage disorder (Fabry's disease, MPS), drugs, radiation, post-cardiac transplantation and genetic. Genetic causes encompasses mutations in sarcomeric (troponin I and T, actin, myosin and titin) and nonsarcomeric protein-coding genes (Desmin, RSK2, lamin A/C and bcl-2-associated athanogene 3 (

Indexed as

CardiomyopathiesCardiomyopathy, RestrictiveAdaptor Proteins, Signal TransducingApoptosis Regulatory ProteinsFilaminsHumansMutationPhenotypeAdaptor Proteins, Signal TransducingApoptosis Regulatory ProteinsBAG3 protein, humanFilaminsFLNC protein, human

Identifiers

PMID36560844
OpenAlexW4312195908

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.