SynthesisNutrients2022
Wernicke Encephalopathy Presenting with Dysphagia: A Case Report and Systematic Literature Review.
Synthesis in Nutrients, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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Who cites it
6 citing papers in PubMed, 10 citations in OpenAlex.
- Case Report: Case of Wernicke's encephalopathy complicated by postoperative gastroparesis following sigmoid colon malignant tumor surgery with a literature review.Frontiers in oncology · 2026Article
- Case Report: Wernicke's encephalopathy induced by prolonged fasting due to apparent psychogenic dysphagia.Frontiers in behavioral neuroscience · 2026Article
- Thiamine Deficiency Secondary to Pain-Induced Oral Aversion.Clinical pediatrics · 2025Article
- Thiamine, gastrointestinal beriberi and acetylcholine signaling.Frontiers in nutrition · 2025Review
- The Role of Nutrition in Neurological Disorders.Nutrients · 2023Article
- Case report: Wernicke-Korsakoff syndrome after bariatric surgery.Frontiers in nutrition · 2023Article
Corrections and comments
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Wernicke encephalopathy (WE) is a well-known neurological condition caused by thiamine (vitamin B1) deficiency that occurs in both alcoholic and non-alcoholic populations. We aimed to report a case of a patient with WE who presented with dysphagia and dysphonia and later developed typical symptoms of thiamine deficiency and to conduct a systematic review of the literature on this rare presentation of WE. We searched two databases (PubMed and Scopus) and included publications up to November 2022. We found 12 cases of WE and dysphagia, aged between 12 and 81 years; swallowing problems presented at the onset in nine patients (including the current case report). Our findings suggest that thiamine deficiency should be suspected in patients with dysphagia of unknown cause, even in the absence of alcohol abuse. In contrast to most WE patients, the majority of patients included in this review presented with dysphagia at the onset of their disease, even in the absence of the classic triad of cognitive impairment, ataxia, and oculomotor abnormalities, indicating that there could be varying susceptibilities to clinical manifestations of thiamine deficiency in different brain regions.
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Registered trials
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