Evidence map›Paper›PMID 36555314›Full record

ArticleInternational journal of molecular sciences2022

Role of EZH2 in Uterine Gland Development.

Nan Ni, Frank L Jalufka, Xin Fang, Dylan A McCreedy, Qinglei Li

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.5field-weighted citation impact, top 37% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Decoding Müllerian Duct Epithelial Regionalization.Molecular reproduction and development · 2025
    Review
  3. Article
  4. Uterine-specificiScience · 2023
    Article
  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Nan NiDepartment of Veterinary Integrative Biosciences, Texas A&M University, College Station, TX 77843, USA.
Frank L JalufkaDepartment of Biology, Texas A&M University, College Station, TX 77843, USA.
Xin FangDepartment of Veterinary Integrative Biosciences, Texas A&M University, College Station, TX 77843, USA.
Dylan A McCreedyDepartment of Biology, Texas A&M University, College Station, TX 77843, USA.
Qinglei LiDepartment of Veterinary Integrative Biosciences, Texas A&M University, College Station, TX 77843, USA.
Texas A&M University · US

Funding

CRR LIGAND ASSAY AND ANALYSIS CORER24HD102061 · NICHD · UNIVERSITY OF VIRGINIA · PI HAISENLEDER, DANIEL J. · 2019 to 2023
$2.2M
Supplement: L-selectin shedding as a novel therapeutic strategy to mitigate acute secondary damage after spinal cord injuryR01NS122961 · NINDS · TEXAS A&M UNIVERSITY · PI MCCREEDY, DYLAN A. · 2021 to 2025
$2.0M
TGF-BETA SIGNALING IN ENDOMETRIAL CELL FUNCTION AND DYSFUNCTIONR01HD087236 · NICHD · TEXAS A&M AGRILIFE RESEARCH · PI LI, QINGLEI · 2017 to 2021
$1.5M
UTX: A novel regulator of decidualization?R03HD107958 · NICHD · TEXAS A&M UNIVERSITY · PI LI, QINGLEI · 2022 to 2023
$146k
NICHD NIH HHS R01 HD087236NICHD NIH HHS R03 HD107958NICHD NIH HHS R24 HD102061NINDS NIH HHS R01 NS122961
6 · The paper itself

Abstract

Enhancer of zeste homolog 2 (EZH2) is a core component of polycomb repressive complex 2 that plays a vital role in transcriptional repression of gene expression. Conditional ablation of EZH2 using progesterone receptor (

Indexed as

Enhancer of Zeste Homolog 2 ProteinUterusAnimalsEpithelial CellsFemaleMiceMice, KnockoutOrganogenesisPregnancyEnhancer of Zeste Homolog 2 ProteinEzh2 protein, mouseadenogenesisendometriumEZH2

Identifiers

PMID36555314
PMCPMC9779349
OpenAlexW4312123481

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.