Evidence map›Paper›PMID 36437534›Full record

ArticleThe Journal of international medical research2022

The metabolic phenotype of the patient influences the reduction in carotid intima-media thickness achieved following metabolic surgery.

Alberto Melchor-López, Juan Antonio Suárez-Cuenca, Diana Zaineff Banderas-Lares, Gustavo De la Peña-Sosa, Moisés Salamanca-García, Eduardo Vera-Gómez, Alejandro Hernández-Patricio, Juan Ariel Gutiérrez-Buendía, Carlos Ramiro Zamora-Alemán, Sofía Lizeth Alcaráz-Estrada and 12 more

Open access · goldAbstract read
In one paragraph

Article in The Journal of international medical research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 45% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

22 authors at 4 institutions in 1 country.

Alberto Melchor-LópezInternal Medicine Department, Hospital General "Xoco" SS CDMX, Mexico City, Mexico.
Juan Antonio Suárez-CuencaInternal Medicine Department, Hospital General "Xoco" SS CDMX, Mexico City, Mexico.ORCID 0000-0002-2098-5658
Diana Zaineff Banderas-LaresLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.
Gustavo De la Peña-SosaLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.ORCID 0000-0002-6350-5845
Moisés Salamanca-GarcíaPathology Department, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Eduardo Vera-GómezLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.
Alejandro Hernández-PatricioLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.
Juan Ariel Gutiérrez-BuendíaLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.
Carlos Ramiro Zamora-AlemánLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.ORCID 0000-0002-0316-9552
Sofía Lizeth Alcaráz-EstradaDivisión de Medicina Genómica, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Moisés Ortiz-FernándezBariatric Surgery Department, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Jesús Montoya-RamírezBariatric Surgery Department, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Omar Felipe Gaytán-FuentesBariatric Surgery Department, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Mónica Escamilla-TilchTissue Engineering & Regenerative Medicine Research Group, Coordinación de Investigación, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Juan Antonio Pineda-JuárezTissue Engineering & Regenerative Medicine Research Group, Coordinación de Investigación, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Mario Antonio Téllez-GonzálezTissue Engineering & Regenerative Medicine Research Group, Coordinación de Investigación, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Martha Eunice Rodríguez-ArellanoLaboratorio de Medicina Genómica, Hospital Regional "Lic, Adolfo López Mateos" ISSSTE, Mexico City, Mexico.
Alejandra Contreras-RamosLaboratorio de Biología del Desarrollo y Teratogénesis Experimental, Hospital Infantil de México Federico Gómez, Mexico City, Mexico.
Rolando Efraín Hernández-MuñozDepartamento de Biología Celular y Desarrollo, Instituto de Fisiología Celular, Universidad Nacional Autónoma de México (UNAM), Coyoacán, Mexico City, Mexico.
José Gutiérrez-Salinas
Silvia GarcíaLaboratory of Experimental Metabolism and Clinical Research, Division of Research, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City. Mexico.
Paul Mondragón-TeránTissue Engineering & Regenerative Medicine Research Group, Coordinación de Investigación, Centro Médico Nacional "20 de Noviembre" ISSSTE, Mexico City, Mexico.
Institute for Social Security and Services for State Workers · MXMexican Social Security Institute · MXHospital Infantil de México Federico Gómez · MXUniversidad Nacional Autónoma de México · MX

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectivesTo determine whether metabolic phenotype is associated with the change in carotid intima-media thickness (CIMT) in patients undergoing bariatric /metabolic surgery (BMS).

methodsWe performed a case-control study of BMS candidates who had metabolically unhealthy obesity (MUO) or metabolically healthy obesity (MHO). We measured the change in CIMT during the 9 months following BMS. The plasma tumor necrosis factor-α, interleukin-1β, adiponectin, leptin, nitric oxide (NO), vascular endothelial growth factor A (VEGF-A), and malondialdehyde concentrations were determined, adipocyte area was measured histologically, and adipose tissue area was estimated using computed tomography.

resultsFifty-six patients (mean age 44.5 years, mean body mass index 44.9 kg/m

conclusionThe metabolic phenotype of patients may influence their change in CIMT following BMS, probably through circulating vasodilatory and pro-inflammatory molecules.

Indexed as

Bariatric SurgeryObesity, Metabolically BenignCarotid Intima-Media ThicknessCase-Control StudiesFemaleHumansMaleObesityRisk FactorsVascular Endothelial Growth Factor AVascular Endothelial Growth Factor Abariatric surgerycarotid intima-media thicknessmetabolic phenotypeMetabolic surgerynitric oxideobesityvascular endothelial growth factor A

Identifiers

PMID36437534
PMCPMC9709181
OpenAlexW4310261371

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.