Evidence map›Paper›PMID 36421822›Full record

ArticleGenes2022

Polymorphic Variants of

Anna Balcerzyk-Matić, Tomasz Nowak, Katarzyna Mizia-Stec, Joanna Iwanicka, Tomasz Iwanicki, Paweł Bańka, Alicja Jarosz, Artur Filipecki, Iwona Żak, Jolanta Krauze and 1 more

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Genes, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.7field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 4 citations in OpenAlex.

  1. Role ofCurrent medicinal chemistry · 2025
    Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 2 institutions in 1 country.

Anna Balcerzyk-MatićDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.ORCID 0000-0002-1113-7796
Tomasz NowakDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.ORCID 0000-0002-6712-5911
Katarzyna Mizia-StecFirst Department of Cardiology, School of Medicine in Katowice, Medical University of Silesia, 47 Ziołowa St., 40-635 Katowice, Poland.
Joanna IwanickaDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.ORCID 0000-0002-3609-9554
Tomasz IwanickiDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.ORCID 0000-0003-0044-3162
Paweł BańkaFirst Department of Cardiology, School of Medicine in Katowice, Medical University of Silesia, 47 Ziołowa St., 40-635 Katowice, Poland.
Alicja JaroszDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.ORCID 0000-0002-8159-6437
Artur FilipeckiFirst Department of Cardiology, School of Medicine in Katowice, Medical University of Silesia, 47 Ziołowa St., 40-635 Katowice, Poland.
Iwona ŻakDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.
Jolanta KrauzeAmerican Heart of Poland, Armii Krajowej 101 Avenue, 43-316 Bielsko-Biała, Poland.
Paweł NiemiecDepartment of Biochemistry and Medical Genetics, School of Health Sciences in Katowice, Medical University of Silesia, Medykow Street 18, 40-752 Katowice, Poland.
Medical University of Silesia · PLAmerican Heart of Poland · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Genetic factors can influence the risk of coronary artery disease (CAD) and the survival of patients. Our previous research led to the identification of genetic variants predisposing to CAD in the Polish population. Since many of them affect the clinical phenotype of the disease, the aim of this study was searching for genetic factors potentially influencing survival in patients with CAD. The study included 276 patients hospitalized due to coronary artery disease. The database of medical history and genotypic results of 29 polymorphisms were used. The endpoint was defined as death from cardiovascular causes. Survival was defined as the period from angiographic confirmation of CAD to death from cardiovascular causes. Three of all the analyzed genes were associated with survival. In the case of the

Indexed as

AngiotensinogenATP Binding Cassette Transporter 1Coronary Artery DiseaseNADPH OxidasesAllelesDisease SusceptibilityGenotypeHumansPolymorphism, GeneticProspective StudiesABCA1 protein, humanAGT protein, humanAngiotensinogenATP Binding Cassette Transporter 1CYBA protein, humanNADPH Oxidasescoronary artery diseasepolymorphismsurvival

Identifiers

PMID36421822
PMCPMC9690336
OpenAlexW4309719912

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.