Evidence map›Paper›PMID 36411793›Full record

ArticleComputational and mathematical methods in medicine2022

In Silico Evaluation of Nonsynonymous SNPs in Human ADAM33: The Most Common Form of Genetic Association to Asthma Susceptibility.

Milad Mohkam, Nasim Golkar, Seyed Hesamodin Nabavizadeh, Hossein Esmaeilzadeh, Aydin Berenjian, Zahra Ghahramani, Ahmad Golami, Soheila Alyasin

Open access · hybridAbstract read
In one paragraph

Article in Computational and mathematical methods in medicine, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.6field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 4 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 2 countries.

Milad MohkamAllergy Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Nasim GolkarPharmaceutical Sciences Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID https://orcid.org/0000-0002-2930-8522
Seyed Hesamodin NabavizadehAllergy Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Hossein EsmaeilzadehAllergy Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Aydin BerenjianSchool of Engineering, University of Waikato, Hamilton 3240, New Zealand.
Zahra GhahramaniSchool of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Ahmad GolamiBiotechnology Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID https://orcid.org/0000-0003-1851-159X
Soheila AlyasinAllergy Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.ORCID https://orcid.org/0000-0001-7183-545X
Shiraz University of Medical Sciences · IRUniversity of Waikato · NZ

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

ADAM33 is a zinc-dependent metalloprotease of the ADAM family, which plays a vital biological role as an activator of Th

Indexed as

AsthmaPolymorphism, Single NucleotideADAM ProteinsCase-Control StudiesGenetic Predisposition to DiseaseHumansADAM33 protein, humanADAM Proteins

Identifiers

PMID36411793
PMCPMC9675607
OpenAlexW4309072593

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.