ArticleNature communications2022
Depletion of CD206
Open access · goldAbstract read
In one paragraph
Article in Nature communications, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 46 papers.
0numbers the graph read from it
0cells of the map it votes in
46citing papers in PubMed
5.3field-weighted citation impact, top 3% of its field
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
46 citing papers in PubMed, 67 citations in OpenAlex.
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- Macrophage regulation of extracellular matrix remodeling in aging skeletal muscle.Ageing research reviews · 2026Review
- Mesenchymal Stromal Cell-Mediated Intercellular Communication: Mapping the Interactome for Skeletal Muscle Homeostasis and Regeneration.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026Article
- Deletion of Tgf-β1 From CD206Journal of cachexia, sarcopenia and muscle · 2026Article
- Multicellular senescence impairs skeletal muscle recovery following disuse in aging.Science advances · 2026Article
- Macrophage and fibro-adipogenic progenitor communication in skeletal muscle regeneration: tissue homeostasis and pathogenic remodeling.Journal of leukocyte biology · 2026Review
- GSDME-IL-18 pyroptotic axis prevents myosteatosis by expanding tissue-resident macrophages to promote muscle regeneration.The Journal of clinical investigation · 2026Article
- Cannabinoid type 2 receptor regulates skeletal muscle regeneration by NLRP3-GSDMD mediated macrophage pyroptosis after injury.Cell death discovery · 2026Article
- Inflammaging-induced TRAF3 degradation impairs AMP biosynthesis to drive sarcopenia.Research square · 2026Article
- RUNX2 Activation in Fibro/Adipogenic Progenitors Promotes Muscle Fibrosis in Muscular Dystrophy.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026Article
- Macrophage-Myocyte Cross Talk Induces M1 Polarization and Inflammatory Cytokine Production During Cancer Cachexia.Mediators of inflammation · 2026Article
- Macrophage plasticity and metabolic control in muscle repair and disease.Frontiers in immunology · 2026Review
- Loss of Lipin1 Contributes to Multiple Pathological Processes in the Development of Heart Failure.Journal of the American Heart Association · 2025Article
- Vitamin B12-Loaded Chitosan Nanoparticles Promote Skeletal Muscle Injury Repair in Aged Rats via Amelioration of Aging-Suppressed Efferocytosis.Biomolecules · 2025Article
- HIV infection and immunosenescence: challenges and intervention strategies.BMC medicine · 2025Review
- Deletion of fibro-adipogenic progenitors-specific follistatin impairs muscle function and accelerates skeletal muscle atrophy in obese mice.Molecular medicine (Cambridge, Mass.) · 2025Article
- Tofogliflozin ameliorates cardiotoxin induced skeletal muscle injury and fibrosis in obesity.Scientific reports · 2025Article
- Article
- A combinatorial oligonucleotide therapy to improve dystrophin restoration and dystrophin-deficient muscle health.Molecular therapy. Nucleic acids · 2025Article
- miRNA-125b-5p-rich exosomes derived from fibro-adipogenic progenitors promoting muscle regeneration through inducing pro-regenerative macrophages.Stem cell research & therapy · 2025Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
24 authors at 7 institutions in 3 countries.
Allah NawazDepartment of Molecular and Medical Pharmacology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan. allah.nawaz@joslin.harvard.edu.ORCID 0000-0002-1979-9575
Muhammad BilalFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Shiho FujisakaFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Tomonobu KadoFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan. kado@med.u-toyama.ac.jp.
Muhammad Rahil AslamFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.ORCID 0000-0003-0684-9147
Saeed AhmedDepartment of Medicine and Surgery, Rawalpindi Medical University, Rawalpindi, Punjab, 46000, Pakistan.
Keisuke OkabeDepartment of Molecular and Medical Pharmacology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Yoshiko IgarashiFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Yoshiyuki WatanabeFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Takahide KuwanoFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Koichi TsuneyamaDepartment of Pathology and Laboratory Medicine, Institute of Biomedical Sciences, Tokushima University Graduate School, 3-18-15 Kuramoto, Tokushima, 770-8503, Japan.
Ayumi NishimuraFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Yasuhiro NishidaFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Seiji YamamotoDepartment of Pathology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.ORCID 0000-0003-1007-6271
Masakiyo SasaharaDepartment of Pathology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Johji ImuraDepartment of Diagnostic Pathology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.
Hisashi MoriDepartment of Molecular Neuroscience, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.ORCID 0000-0001-9743-2456
Martin M MatzukDepartment of Pathology and Immunology, Baylor College of Medicine, Houston, TX, 77030-3411, USA.
Fujimi KudoDepartment of Systems Medicine, Chiba University Graduate School of Medicine, 1-8-1, Inohana, Chuo-ku, Chiba, 260-8670, Japan.
Ichiro ManabeDepartment of Systems Medicine, Chiba University Graduate School of Medicine, 1-8-1, Inohana, Chuo-ku, Chiba, 260-8670, Japan.
Akiyoshi UezumiDepartment of Nutritional Physiology, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, Tokushima, 770-8503, Japan.
Takashi NakagawaDepartment of Molecular and Medical Pharmacology, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan.ORCID 0000-0001-5539-7605
Yumiko OishiDepartment of Biochemistry and Molecular Biology, Nippon Medical School, 1-1-5 Sendagi, Bunkyo-ku, Tokyo, 113-8602, Japan.
Kazuyuki TobeFirst Department of Internal Medicine, Faculty of Medicine, University of Toyama, Toyama-shi, Toyama, 930-0194, Japan. tobe@med.u-toyama.ac.jp.
University of Toyama · JPChiba University · JPTokushima University · JPBaylor College of Medicine · USJoslin Diabetes Center · USNippon Medical School · JPRawalpindi Medical University · PK
Funding
No grant is acknowledged in the PubMed record.
Abstract
Muscle regeneration requires the coordination of muscle stem cells, mesenchymal fibro-adipogenic progenitors (FAPs), and macrophages. How macrophages regulate the paracrine secretion of FAPs during the recovery process remains elusive. Herein, we systemically investigated the communication between CD206
Indexed as
AdipogenesisFollistatinAnimalsMacrophagesMannose ReceptorMiceMice, TransgenicMusclesFollistatinMannose Receptor
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.