Evidence map›Paper›PMID 36405232›Full record

ReviewCardiology research2022

Is Long COVID Syndrome a Transient Mitochondriopathy Newly Discovered: Implications of CPET.

Stefanos G Sakellaropoulos, Muhammad Ali, Athanasios Papadis, Muhemin Mohammed, Andreas Mitsis, Zaza Zivzivadze

Abstract readReview
In one paragraph

Review in Cardiology research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Stefanos G SakellaropoulosDepartment of Internal Medicine, Cardiology, Cantonal Hospital Baden, Baden, Switzerland.
Muhammad AliClinic of Cardiology Salzgitter, Salzgitter, Germany.
Athanasios PapadisSwiss Cardiovascular Centre, Bern University Hospital, Bern, Switzerland.
Muhemin MohammedClinic of Internal Medicine, Hospital Affoltern, Affoltern, Switzerland.
Andreas MitsisCardiology Department, Nicosia General Hospital, Nicosia, Cyprus.
Zaza ZivzivadzeDepartment of Emergency Care, Hospital Affoltern, Affoltern, Switzerland.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The new outbreak of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection has an impact worldwide, primarily as a medical emergency. Even that the total constellation is considered as a pandemic of acute respiratory disease, coronavirus disease 2019 (COVID-19) gives rise to dysfunction in multiple organs (e.g., brain, lungs, heart, muscles) that impairs cardiopulmonary (CP) function. Parallel to the CP consequences of COVID-19 is a significant reduction in physical activity. Cardiopulmonary exercise testing (CPET) is daily used in clinical practice to define prognosis, provide risk stratification and treatment strategy. As such, the significance of CPET is crucial concerning clinical assessments of COVID-19 patients. Furthermore, new studies aim at understanding the effects of SARS-CoV-2 infection in long term. Multiple studies have investigated the cardiopulmonary function and impairment of exercise endurance in such patients, as well as a possible prolonged physical impairment. With this review, we summarize the COVID-19-associated pathophysiology for the Long COVID (LC) syndrome as well as the importance of performing CPET.

Indexed as

Cardiopulmonary exercise testLong COVID syndromeMitochondriopathy

Identifiers

PMID36405232
PMCPMC9635775

What OpenQuestion holds

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LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.