Evidence map›Paper›PMID 36369640›Full record

ArticleJournal of applied genetics2023

Molecular background of Leber congenital amaurosis in a Polish cohort of patients-novel variants discovered by NGS.

Anna Skorczyk-Werner, Anna Sowińska-Seidler, Anna Wawrocka, Joanna Walczak-Sztulpa, Maciej Robert Krawczyński

Open access · hybridAbstract read
In one paragraph

Article in Journal of applied genetics, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.6field-weighted citation impact, top 35% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 8 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Anna Skorczyk-WernerDepartment of Medical Genetics, Poznan University of Medical Sciences, Poznan, Poland. aniaskorczyk@poczta.onet.pl.ORCID http://orcid.org/0000-0002-8847-1776
Anna Sowińska-SeidlerDepartment of Medical Genetics, Poznan University of Medical Sciences, Poznan, Poland.
Anna WawrockaDepartment of Medical Genetics, Poznan University of Medical Sciences, Poznan, Poland.
Joanna Walczak-SztulpaDepartment of Medical Genetics, Poznan University of Medical Sciences, Poznan, Poland.
Maciej Robert KrawczyńskiDepartment of Medical Genetics, Poznan University of Medical Sciences, Poznan, Poland.
Poznan University of Medical Sciences · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Leber congenital amaurosis (LCA) is the most severe form of inherited retinal dystrophies and the most frequent cause of congenital blindness in children. To date, 25 genes have been implicated in the pathogenesis of this rare disorder. Performing an accurate molecular diagnosis is crucial as gene therapy is becoming available. This study aimed to report the molecular basis of Leber congenital amaurosis, especially novel and rare variants in 27 Polish families with a clinical diagnosis of LCA fully confirmed by molecular analyses. Whole exome sequencing or targeted next-generation sequencing (NGS) of inherited retinal dystrophies-associated (IRD) genes was applied to identify potentially pathogenic variants. Bidirectional Sanger sequencing and quantitative PCR (qPCR) were carried out for validation and segregation analysis of the variants identified within the families. We identified 28 potentially pathogenic variants, including 11 novel, in 8 LCA genes: CEP290, CRB1, GUCY2D, NMNAT1, RPGRIP1, CRX, LRAT1, and LCA5. This study expands the mutational spectrum of the LCA genes. Moreover, these results, together with the conclusions from our previous studies, allow us to point to the most frequently mutated genes and variants in the Polish cohort of LCA patients.

Indexed as

Leber Congenital AmaurosisNicotinamide-Nucleotide AdenylyltransferaseRetinal DystrophiesAntigens, NeoplasmCell Cycle ProteinsChildCytoskeletal ProteinsDNA Mutational AnalysisEye ProteinsHigh-Throughput Nucleotide SequencingHumansMembrane ProteinsMutationNerve Tissue ProteinsPedigreePolandAntigens, NeoplasmCell Cycle ProteinsCep290 protein, humanCRB1 protein, humanCytoskeletal ProteinsEye ProteinsMembrane ProteinsNerve Tissue ProteinsNicotinamide-Nucleotide AdenylyltransferaseNMNAT1 protein, humanInherited retinal dystrophies (IRDs)Leber congenital amaurosis (LCA)Novel variantsTargeted next-generation sequencing (NGS)Whole exome sequencing (WES)

Identifiers

PMID36369640
PMCPMC9837007
OpenAlexW4308834768

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.