Evidence map›Paper›PMID 36348176›Full record

ArticleEuropean archives of paediatric dentistry : official journal of the European Academy of Paediatric Dentistry2023

Molar-incisor hypomineralisation prevalence in a cohort of Australian children with type 1 diabetes.

C Lim, E D Jensen, B F Poirier, S Sethi, G Smart, A S Peña

Open access · hybridAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in European archives of paediatric dentistry : official journal of the European Academy of Paediatric Dentistry, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.3field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

C LimAdelaide Dental School, University of Adelaide, 4 North Terrace, Adelaide, SA, 5000, Australia.
E D JensenAdelaide Dental School, University of Adelaide, 4 North Terrace, Adelaide, SA, 5000, Australia. emilija.jensen@adelaide.edu.au.ORCID http://orcid.org/0000-0001-9462-2596
B F PoirierAdelaide Dental School, University of Adelaide, 4 North Terrace, Adelaide, SA, 5000, Australia.
S SethiAdelaide Dental School, University of Adelaide, 4 North Terrace, Adelaide, SA, 5000, Australia.
G SmartDepartment of Paediatric Dentistry, Women's and Children's Hospital, Adelaide, SA, Australia.
A S PeñaDiscipline of Paediatrics and Robinson Research Institute, University of Adelaide, Adelaide, SA, Australia.
The University of Adelaide · AUWomen's and Children's Hospital · AU

Funding

Women's and Children's Hospital Foundation G0077SWomen's and Children's Hospital Foundation G00801
6 · The paper itself

Abstract

purposeSystemic diseases or drugs administered early in life may cause a disruption in amelogenesis and contribute to the qualitative defect of enamel described as molar-incisor hypomineralisation (MIH). Therefore, an increase in prevalence of MIH in children with type 1 diabetes (T1D) may be expected as this systemic disorder is commonly diagnosed in early childhood. The aim of this study was to determine the prevalence of MIH in a cohort of children with T1D and investigate diagnosis of MIH with T1D factors.

methodsCross-sectional study of children with T1D recruited from paediatric diabetes clinics at the Women's and Children's Hospital (South Australia). A detailed medical history, comprehensive dental and MIH examination according to the European Academy of Paediatric Dentistry (EAPD) long form classification was collected for each child. All upper and lower first permanent molars and central incisors were scored.

resultsA total number of 73 participants; 35 (47.95%) males were examined including 584 teeth. The mean age of the participants was 13.25 ± 2.58 years, with a mean age of diagnosis 7.75 ± 3.58 years, and a mean HbA1c of 8.5 ± 1.6%. 42 out of 73 children (54.8%) had enamel defects on at least one of the teeth examined. However, 19.2% met the criteria for MIH. Univariate and bivariate analyses were conducted but no significant associations were noted between MIH and risk factors including diabetes control (p > 0.1).

conclusionThere was a high prevalence of enamel defects and MIH amongst children with T1D. More research is required to establish association between T1D and MIH.

Indexed as

Dental Enamel HypoplasiaDiabetes Mellitus, Type 1Molar HypomineralizationAdolescentAustraliaChildChild, PreschoolCross-Sectional StudiesFemaleHumansMaleMolarPrevalenceChildrenMIHMolar-incisor hypomineralisationT1DType 1 diabetes

Identifiers

PMID36348176
PMCPMC9992226
OpenAlexW4308531263

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.