Evidence map›Paper›PMID 36320442›Full record

ReviewJournal of the renin-angiotensin-aldosterone system : JRAAS2022

View of the Renin-Angiotensin System in Acute Kidney Injury Induced by Renal Ischemia-Reperfusion Injury.

Farzaneh Karimi, Maryam Maleki, Mehdi Nematbakhsh

Abstract readReview
In one paragraph

Review in Journal of the renin-angiotensin-aldosterone system : JRAAS, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

  1. Trial
  2. Review
  3. Article
  4. Review
  5. Observational
  6. Article
  7. Article
  8. Article
  9. Role of G protein coupled receptors in acute kidney injury.Cell communication and signaling : CCS · 2024
    Review
  10. Article
  11. Review
  12. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Farzaneh KarimiDepartment of Physiology, Behbahan Faculty of Medical Sciences, Behbahan, Iran.ORCID https://orcid.org/0000-0002-1512-7333
Maryam MalekiDepartment of Physiology, Faculty of Medicine, Ilam University of Medical Sciences, Ilam, Iran.ORCID https://orcid.org/0000-0002-1342-553X
Mehdi NematbakhshWater & Electrolytes Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.ORCID https://orcid.org/0000-0002-9565-9187

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Renal ischemia-reperfusion injury (RIRI) is a sequence of complicated events that is defined as a reduction of the blood supply followed by reperfusion. RIRI is the leading cause of acute kidney injury (AKI). Among the diverse mediators that take part in RIRI-induced AKI, the renin-angiotensin system (RAS) plays an important role via conventional (angiotensinogen, renin, angiotensin-converting enzyme (ACE), angiotensin (Ang) II, and Ang II type 1 receptor (AT

Indexed as

Acute Kidney InjuryReperfusion InjuryAngiotensin-Converting Enzyme 2Angiotensin IIHumansKidneyPeptidyl-Dipeptidase ARenin-Angiotensin SystemAngiotensin-Converting Enzyme 2Angiotensin IIPeptidyl-Dipeptidase A

Identifiers

PMID36320442
PMCPMC9617735

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.