Evidence map›Paper›PMID 36293477›Full record

ReviewInternational journal of molecular sciences2022

Epigenetic Changes in Prion and Prion-like Neurodegenerative Diseases: Recent Advances, Potential as Biomarkers, and Future Perspectives.

Adelaida Hernaiz, Janne Markus Toivonen, Rosa Bolea, Inmaculada Martín-Burriel

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.7field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 21 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Review
  6. Special Issue "Epigenetics in Neurodegenerative Diseases".International journal of molecular sciences · 2024
    Article
  7. Article
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Adelaida HernaizLaboratorio de Genética Bioquímica (LAGENBIO), Facultad de Veterinaria, Universidad de Zaragoza, IA2, IIS Aragón, 50013 Zaragoza, Spain.ORCID 0000-0002-6806-9990
Janne Markus ToivonenLaboratorio de Genética Bioquímica (LAGENBIO), Facultad de Veterinaria, Universidad de Zaragoza, IA2, IIS Aragón, 50013 Zaragoza, Spain.ORCID 0000-0002-7243-1737
Rosa BoleaCentro de Encefalopatías y Enfermedades Transmisibles Emergentes (CEETE), Facultad de Veterinaria, Universidad de Zaragoza, IA2, IIS Aragón, 50013 Zaragoza, Spain.
Inmaculada Martín-BurrielLaboratorio de Genética Bioquímica (LAGENBIO), Facultad de Veterinaria, Universidad de Zaragoza, IA2, IIS Aragón, 50013 Zaragoza, Spain.ORCID 0000-0001-6016-4726
Universidad de Zaragoza · ES

Funding

Fondo Europeo de Desarrollo Regional (FEDER) A19-20RGobierno de Aragón A19-20RGobierno de Aragón IIU/2023/2017Ministry of Economy, Industry and Competitiveness AGL2015-67945-P
6 · The paper itself

Abstract

Prion diseases are transmissible spongiform encephalopathies (TSEs) caused by a conformational conversion of the native cellular prion protein (PrP

Indexed as

MicroRNAsNeurodegenerative DiseasesPrion DiseasesPrionsBiomarkersEpigenesis, GeneticHistonesHumansPrion ProteinsBiomarkersHistonesMicroRNAsPrion ProteinsPrionsDNA methylationepigeneticshistone modificationsmicroRNAprion diseasesprion-like diseases

Identifiers

PMID36293477
PMCPMC9604074
OpenAlexW4306925904

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.