Evidence map›Paper›PMID 36278864›Full record

ArticleeLife2022

CD169

Jamie Moore-Fried, Mahinder Paul, Zhixin Jing, David Fooksman, Gregoire Lauvau

Open access · goldAbstract read
In one paragraph

Article in eLife, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.5field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 7 citations in OpenAlex.

  1. CD169Inflammation research : official journal of the European Histamine Research Society ... [et al.] · 2026
    Article
  2. Review
  3. Exploring CD169Vaccines · 2025
    Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Jamie Moore-Fried *Albert Einstein College of Medicine, Department of Microbiology and Immunology, Bronx, United States.
Mahinder Paul *Albert Einstein College of Medicine, Department of Microbiology and Immunology, Bronx, United States.
Zhixin JingAlbert Einstein College of Medicine, Department of Microbiology and Immunology, Bronx, United States.
David FooksmanAlbert Einstein College of Medicine, Department of Microbiology and Immunology, Bronx, United States.
Gregoire LauvauAlbert Einstein College of Medicine, Department of Microbiology and Immunology, Bronx, United States.ORCID 0000-0002-3050-2664
Albert Einstein College of Medicine · US

Funding

MEDICAL SCIENTIST TRAINING PROGRAMT32GM007288 · NIGMS · YESHIVA UNIVERSITY · PI AKABAS, MYLES H. · 1985 to 2022
$35.6M
Mechanisms of Plasmacytoid dendritic cell activation in vivoR21AI128735 · NIAID · ALBERT EINSTEIN COLLEGE OF MEDICINE, INC · PI FOOKSMAN, DAVID R, LAUVAU, GREGOIRE STEPHANE · 2017 to 2018
$459k
Unraveling Mechanisms of Plasmacytoid Dendritic Cell Priming by CD169+ Macrophages in Severe Murine MalariaF31HL147470 · NHLBI · ALBERT EINSTEIN COLLEGE OF MEDICINE · PI MOORE, JAMIE MARIE · 2019 to 2019
$50k
NHLBI NIH HHS F31 HL147470NIAID NIH HHS R21 AI128735NIGMS NIH HHS T32 GM007288NIH HHS F31 HL147470
6 · The paper itself

Abstract

Plasmacytoid dendritic cells (pDCs) are the most potent producer of type I interferon (IFN), but how pDC is primed in vivo is poorly defined. Using a mouse model of severe malaria, we have previously established that upon priming by CD169

Indexed as

Bone MarrowDendritic CellsMacrophagesMalariaToll-Like Receptor 7AnimalsMiceToll-Like Receptor 7bone marrowCD169+ macrophageimmunologyinfectious diseaseinflammationintravital imagingmicrobiologymouseplasmacytoid dendritic cellplasmodiumsevere malariaSTINGtype I interferon

Identifiers

PMID36278864
PMCPMC9648966
OpenAlexW4307180644

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.