Evidence map›Paper›PMID 36267691›Full record

ArticleInfectious Disease Modelling2022

The role of super-spreaders in modeling of SARS-CoV-2.

François Rousse, Marcus Carlsson, Magnus Ögren, Benjamin Kalischer Wellander

Abstract read
In one paragraph

Article in Infectious Disease Modelling, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

François RousseSchool of Science and Technology, Örebro University, 70182, Örebro, Sweden.
Marcus CarlssonCentre for Mathematical Sciences, Lund University, Box 118, 22100, Lund, Sweden.
Magnus ÖgrenSchool of Science and Technology, Örebro University, 70182, Örebro, Sweden.
Benjamin Kalischer WellanderCentrum för forskning och utveckling (CFUG) Region Gävleborg, Sweden.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In stochastic modeling of infectious diseases, it has been established that variations in infectivity affect the probability of a major outbreak, but not the shape of the curves during a major outbreak, which is predicted by deterministic models (Diekmann et al., 2012). However, such conclusions are derived under idealized assumptions such as the population size tending to infinity, and the individual degree of infectivity only depending on variations in the infectiousness period. In this paper we show that the same conclusions hold true in a finite population representing a medium size city, where the degree of infectivity is determined by the offspring distribution, which we try to make as realistic as possible for SARS-CoV-2. In particular, we consider distributions with fat tails, to incorporate the existence of super-spreaders. We also provide new theoretical results on convergence of stochastic models which allows to incorporate any offspring distribution with a finite variance.

Indexed as

Compartmental modelsCOVID-19Offspring distribution for SARS-CoV-2SEIRSIR

Identifiers

PMID36267691
PMCPMC9558769

What OpenQuestion holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.