ArticleLaryngoscope investigative otolaryngology2022
Demographic and pathologic factor regression to a growth rate model of p16-negative oral cavity squamous cell carcinoma.
Article in Laryngoscope investigative otolaryngology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
3 citing papers in PubMed, 2 citations in OpenAlex.
- Tumor Growth Rate in Head and Neck Squamous Cell Carcinoma: A Scoping Review.Laryngoscope investigative otolaryngology · 2026Review
- Do we recognize oral cancer? Primary professional delay in diagnosis of oral squamous cell carcinoma.Clinical oral investigations · 2024Article
- Demographic and pathologic factor regression to a growth rate model of p16-negative oral cavity squamous cell carcinoma.Laryngoscope investigative otolaryngology · 2022Article
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Authors and funding
7 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Objectives: The current study aims to quantify the growth rate of p16-negative oral cavity squamous cell carcinoma, characterize causative relationships between demographic risk factors and tumor growth, and examine pathologic findings associated with the tumor growth rate at a tertiary care institution. It is hypothesized that causative relationships will be drawn between the individual sociodemographic and pathologic factors and oral cavity p16-negative squamous cell carcinoma growth rate. Methods: Prospectively recruited participants, receiving surgical intervention only, were followed from initial staging CT scan to surgical resection. Interval growth was calculated in cm Results: Summary statistics revealed a mean growth rate for the study sample of 1.385cm Conclusions: Through a small prospective cohort sample, the current study suggests clinical associations between alcohol consumption, origination at the retromolar trigone, and clinical nodal stage with rate of tumor growth. Future work will validate these relationships in a larger patient cohort, and against stronger modeling techniques. Level of Evidence: Prospective non-random cohort design.
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