ArticleAnimal reproduction
Genetic control of reproduction in dairy cows under grazing conditions.
Article in Animal reproduction. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
0 citing papers in PubMed, 7 citations in OpenAlex.
No citing paper in PubMed yet.
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Fertility performance is a key driver of the efficiency and profitability of seasonal-calving pasture- based systems of milk production. Since the 1990's and early 2000's, most countries have placed varying levels of emphasis on fertility and survivability traits, and phenotypic performance has started to improve. In recent years, the underlying physiological mechanisms responsible for good or poor phenotypic fertility have started to be unravelled. It is apparent that poor genetic merit for fertility traits is associated with multiple defects across a range of organs and tissues that are antagonistic to achieving satisfactory fertility performance. The principal defects include excessive mobilisation of body condition score (BCS), unfavourable metabolic status, delayed resumption of cyclicity, increased incidence of endometritis, dysfunctional estrous expression, and inadequate luteal phase progesterone concentrations. At a tissue level, coordinated changes in gene expression in different tissues have been observed to orchestrate more favourable BCS, uterine environment and corpus luteum function. Interestingly, cows with poor genetic merit for fertility traits have up-regulated inflammation and immune response pathways in multiple tissues. Sire genetic merit for daughter fertility traits is improving rapidly in the dairy breeds, especially in the predominant Holstein and Friesian breeds. With advances in animal breeding, especially genomic technologies to identify superior sires, genetic merit for fertility traits can be improved much more quickly than they initially declined.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.