Evidence map›Paper›PMID 36246398›Full record

ArticleOxidative medicine and cellular longevity2022

PCAF Accelerates Vascular Senescence via the Hippo Signaling Pathway.

Chaohua Kong, Dongchen Wang, Feng Wang, Yifei Lv, Wenying Zhou, Peng Ye, Yue Gu, Xiaomin Jiang, Linlin Zhu, Zhen Ge and 2 more

Open access · hybridAbstract read
In one paragraph

Article in Oxidative medicine and cellular longevity, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.9field-weighted citation impact, top 30% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 8 citations in OpenAlex.

  1. Article
  2. Targeting the Epigenetic Remodeler GCN5 Prevents Vascular Oxidative Stress and Endothelial Dysfunction in Obesity: Insights in Patients with Cardiometabolic Disease.High blood pressure & cardiovascular prevention : the official journal of the Italian Society of Hypertension · 2025
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 1 institution in 1 country.

Chaohua KongDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.ORCID https://orcid.org/0000-0001-7854-0718
Dongchen WangDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Feng WangDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Yifei LvDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Wenying ZhouDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Peng YeDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Yue GuDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Xiaomin JiangDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.
Linlin ZhuDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.ORCID https://orcid.org/0000-0002-9677-0004
Zhen GeDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.ORCID https://orcid.org/0000-0003-0423-3664
Yuelin ChaoDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.ORCID https://orcid.org/0000-0001-7086-6414
Shaoliang ChenDepartment of Cardiology, Nanjing First Hospital, Nanjing Medical University, Nanjing, China.ORCID https://orcid.org/0000-0002-3887-952X
Nanjing Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

P300/CBP-Associated Factor (PCAF), one of the histone acetyltransferases (HATs), is known to be involved in cell growth and/or differentiation. PCAF is reported to be involved in atherosclerotic plaques and neointimal formation. However, its role in cellular senescence remains undefined. We investigated the potential mechanism for PCAF-mediated cellular senescence. Immunohistochemical (IHC) analysis showed PCAF was distinctly increased in the endothelia of aorta in aged mice. Palmitate acid (PA) or X radiation significantly induced the expression of senescence-associated markers and PCAF in human umbilical vein endothelial cells (HUVECs). PCAF silence in PA-treated HUVECs significantly rescued senescence-associated phenotypes, while PCAF overexpression accelerated it. Additionally, our results showed that Yes1 Associated Transcriptional Regulator (YAP) that acts as end effector of the Hippo signaling pathway is crucial in PCAF-mediated endothelial senescence. YAP activity declining was observed in aged vascular endothelia. Overexpression of YAP partially ameliorated PCAF-induced endothelial senescence. In vivo, endothelial-(EC-) specific PCAF downregulation in aged mice using adeno-associated virus revealed less vascular senescence-associated phenotypes. These results suggested that PCAF mediated endothelial senescence through the Hippo signaling pathway, implying that PCAF may become a potential target for the prevention and treatment of vascular aging.

Indexed as

Endothelial CellsHippo Signaling PathwayAgedAnimalsCellular SenescenceHistone AcetyltransferasesHumansMicep300-CBP-Associated FactorPalmitatesHistone Acetyltransferasesp300-CBP-Associated FactorPalmitates

Identifiers

PMID36246398
PMCPMC9560818
OpenAlexW4302760206

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.