Evidence map›Paper›PMID 36204832›Full record

ReviewNeural regeneration research2023

Potential physiological and pathological roles for axonal ryanodine receptors.

David P Stirling

Open access · goldAbstract readReview
In one paragraph

Review in Neural regeneration research, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.3field-weighted citation impact, top 51% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 3 citations in OpenAlex.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

David P StirlingKentucky Spinal Cord Injury Research Center and Departments of Neurological Surgery, Anatomical Sciences and Neurobiology, Microbiology and Immunology, University of Louisville, School of Medicine, Louisville, KY, USA.
University of Louisville · US

Funding

ER Ca2+ store mediated white matter injuryR01NS092680 · NINDS · UNIVERSITY OF LOUISVILLE · PI STIRLING, DAVID PAUL · 2017 to 2021
$1.9M
NINDS NIH HHS R01 NS092680
6 · The paper itself

Abstract

Clinical disability following trauma or disease to the spinal cord often involves the loss of vital white matter elements including axons and glia. Although excessive Ca

Indexed as

axomyelinic synapseaxonaxoplasmic reticulumcalciumryanodine receptorsecondary axonal degenerationspinal cord injuryvoltage-gated calcium channelwhite matter injury

Identifiers

PMID36204832
PMCPMC9700104
OpenAlexW4296955460

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.