Evidence map›Paper›PMID 36192605›Full record

Trial reportHormones (Athens, Greece)2022

High-intensity interval training ameliorates endothelial dysfunction through adropin, nitric oxide, MR-proADM, and copeptin changes in overweight subjects.

Sadegh Abbasian, Ali Asghar Ravasi, Rahman Soori, Suna Aydin, Sirous Choobineh, Suleyman Aydin

Abstract readRandomized Controlled Trial
PubMed Publisher
In one paragraph

Trial report in Hormones (Athens, Greece), 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed, 1 pooled it
1.3field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 1 synthesis or guideline pooled it, 8 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Unveiling the multifaceted role of adropin in various diseases (Review).International journal of molecular medicine · 2024
    Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 4 institutions in 2 countries.

Sadegh AbbasianDepartment of Exercise Physiology, Sport Sciences Group, Khavaran Institute of Higher Education, Mashhad, Iran.
Ali Asghar RavasiFaculty of Physical Education and Sport Sciences, University of Tehran, Tehran, Iran.
Rahman SooriFaculty of Physical Education and Sport Sciences, University of Tehran, Tehran, Iran.
Suna AydinDepartment of Cardiovascular Surgery, Elazig Fethi Sekin City Hospital, Health Science University, Elazig Campus, Elazig, Turkey.
Sirous ChoobinehFaculty of Physical Education and Sport Sciences, University of Tehran, Tehran, Iran.
Suleyman AydinDepartment of Medical Biochemistry and Clinical Biochemistry (Firat Hormones Research Group), Medical School, Firat University, Elazig, Turkey. saydin1@hotmail.com.
University of Tehran · IRFırat University · TRSağlık Bilimleri Üniversitesi · TRTabaran Institute of Higher Education · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purposeThe purpose of this study was to determine adropin, NO, MR-proADM, and copeptin changes following four different types of high-intensity interval training (HIIT) in men with overweight.

methodsIn the current study, 45 overweight participants were included in the pre-intervention assessments and randomly assigned to the following groups: (1) control, (2) HIIT bike, (3) HIIT short-treadmill, and (4) HIIT long-treadmill groups. The participants were given 10-min sessions of HIIT intervention between 85 and 95% of VO

resultsThere was a statistically significant elevation in adropin and NO levels (p < 0.05), while MR-proADM and copeptin were notably more decreased than those of the control group following the 8 weeks of HIIT interventions (p < 0.01). However, no statistically significant decrease was observed in carotid/femoral intima-media thickness (c/f-IMT) values following the 8-week HIIT interventions, while statistically significant reductions were demonstrated in participants who had no atherosclerotic plaque or IMT < 0.9 mm (p < 0.05).

conclusionsIn conclusion, HIIT had a greater effect on IMT remodeling of the femoral artery than of the carotid artery. Decreased MR-proADM and copeptin and increased adropin levels might act as a physiological surrogate of endothelial dysfunction through increased NO-related signaling pathways in participants with overweight following high-intensity interval training.

Indexed as

High-Intensity Interval TrainingOverweightCarotid Intima-Media ThicknessGlycopeptidesHumansMaleNitric OxidecopeptinsGlycopeptidesNitric OxideAdropinExercise trainingIntima-media thicknessVascular functional surrogates

Identifiers

PMID36192605
OpenAlexW4301391302

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.