Evidence map›Paper›PMID 36043704›Full record

ArticleJournal of neurophysiology2022

Na

Khrystyna Yu Sukhanova, Ankeeta Koirala, Keith S Elmslie

Open access · greenAbstract read
In one paragraph

Article in Journal of neurophysiology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.3field-weighted citation impact, top 44% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 4 citations in OpenAlex.

  1. Article
  2. Role of NaThe Journal of physiology · 2023
    Article
  3. Bradykinin 2 receptors contribute to the exaggerated exercise pressor reflex in a rat model of simulated peripheral artery disease.American journal of physiology. Regulatory, integrative and comparative physiology · 2023
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Khrystyna Yu SukhanovaThe Baker Laboratory of Pharmacology, Department of Pharmacology, Kirksville College of Osteopathic Medicine, A.T. Still University of Health Sciences, Kirksville, Missouri.
Ankeeta KoiralaThe Baker Laboratory of Pharmacology, Department of Pharmacology, Kirksville College of Osteopathic Medicine, A.T. Still University of Health Sciences, Kirksville, Missouri.
Keith S ElmslieThe Baker Laboratory of Pharmacology, Department of Pharmacology, Kirksville College of Osteopathic Medicine, A.T. Still University of Health Sciences, Kirksville, Missouri.ORCID 0000-0002-2988-4482
A.T. Still University · US

Funding

HHS | NIH | National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) AR05976
6 · The paper itself

Abstract

Skeletal muscle contraction triggers the exercise pressor reflex (EPR) to regulate the cardiovascular system response to exercise. During muscle contraction, substances are released that generate action potential activity in group III and IV afferents that mediate the EPR. Some of these substances increase afferent activity via G-protein-coupled receptor (GPCR) activation, but the mechanisms are incompletely understood. We were interested in determining if tetrodotoxin-resistant (TTX-R) voltage-dependent sodium channels (Na

Indexed as

BradykininGanglia, SpinalAdenosine TriphosphateHumansMusclesNAV1.9 Voltage-Gated Sodium ChannelNeurons, AfferentNorepinephrineProstaglandinsSodium Channel BlockersSodium ChannelsTetrodotoxinAdenosine TriphosphateBradykininNAV1.9 Voltage-Gated Sodium ChannelNorepinephrineProstaglandinsSodium Channel BlockersSodium ChannelsTetrodotoxinATPbradykininexercise pressor reflexnorepinephrineprostaglandin E2

Identifiers

PMID36043704
PMCPMC9512110
OpenAlexW4293762571

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.