Evidence map›Paper›PMID 35986126›Full record

ArticleGeroScience2023

Tissue-specific reductions in mitochondrial efficiency and increased ROS release rates during ageing in zebra finches, Taeniopygia guttata.

Pablo Salmón, Caroline Millet, Colin Selman, Pat Monaghan, Neal J Dawson

Open access · hybridAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in GeroScience, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.0field-weighted citation impact, top 24% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 7 citations in OpenAlex.

  1. Review
  2. Understanding age and society using natural populations.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2024
    Article
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Pablo SalmónSchool of Biodiversity, One Health and Veterinary Medicine, University of Glasgow, Graham Kerr Building, Glasgow, G12 8QQ, UK. pablo.salmon@glasgow.ac.uk.ORCID 0000-0001-9718-6611
Caroline MilletSchool of Biodiversity, One Health and Veterinary Medicine, University of Glasgow, Graham Kerr Building, Glasgow, G12 8QQ, UK.
Colin SelmanSchool of Biodiversity, One Health and Veterinary Medicine, University of Glasgow, Graham Kerr Building, Glasgow, G12 8QQ, UK.
Pat MonaghanSchool of Biodiversity, One Health and Veterinary Medicine, University of Glasgow, Graham Kerr Building, Glasgow, G12 8QQ, UK.
Neal J DawsonSchool of Biodiversity, One Health and Veterinary Medicine, University of Glasgow, Graham Kerr Building, Glasgow, G12 8QQ, UK. neal.dawson@glasgow.ac.uk.ORCID 0000-0001-5389-8692
University of Glasgow · GB

Funding

Biotechnology and Biological Sciences Research Council BB/P009174/1Wellcome TrustWellcome Trust 310331-01
6 · The paper itself

Abstract

Mitochondrial dysfunction and oxidative damage have long been suggested as critically important mechanisms underlying the ageing process in animals. However, conflicting data exist on whether this involves increased production of mitochondrial reactive oxygen species (ROS) during ageing. We employed high-resolution respirometry and fluorometry on flight muscle (pectoralis major) and liver mitochondria to simultaneously examine mitochondrial function and ROS (H

Indexed as

FinchesHydrogen PeroxideAgingAnimalsMitochondriaReactive Oxygen SpeciesHydrogen PeroxideReactive Oxygen SpeciesAgeingBirdsMitochondrial physiologyProton leakReactive oxygen speciesSenescence

Identifiers

PMID35986126
PMCPMC9886749
OpenAlexW4292366914

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.