Evidence map›Paper›PMID 35909537›Full record

ArticleFrontiers in endocrinology2022

miR-199a Is Upregulated in GDM Targeting the MeCP2-Trpc3 Pathway.

Chun-Yi Guan, Jing-Li Cao, Lu Zhang, Xue-Qin Wang, Xu Ma, Hong-Fei Xia

Open access · goldAbstract read
In one paragraph

Article in Frontiers in endocrinology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. Review
  3. The role of microRNAs in pregnancies complicated by maternal diabetes.Clinical science (London, England : 1979) · 2024
    Review
  4. Review
  5. Targeting miR-199a reduces fibrosis in hypertrophic cardiomyopathy.Journal of molecular and cellular cardiology plus · 2024
    Article
  6. Review
  7. Review
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Chun-Yi GuanReproductive and Genetic Center of National Research Institute for Family Planning, Beijing, China.
Jing-Li CaoGraduate School, Peking Union Medical College, Beijing, China.
Lu ZhangReproductive and Genetic Center of National Research Institute for Family Planning, Beijing, China.
Xue-Qin WangGraduate School, Peking Union Medical College, Beijing, China.
Xu MaReproductive and Genetic Center of National Research Institute for Family Planning, Beijing, China.
Hong-Fei XiaReproductive and Genetic Center of National Research Institute for Family Planning, Beijing, China.
Peking Union Medical College Hospital · CNChinese Academy of Medical Sciences & Peking Union Medical College · CNNational Health and Family Planning Commission · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Gestational diabetes mellitus (GDM), the most common medical pregnancy complication, has become a growing problem. More and more studies have shown that microRNAs are closely related to metabolic processes. The purpose of this paper is to investigate the role of up-regulation of miR-199a-5p expression in GDM. We found that miR-199a-5p was significantly up-regulated in the placenta of GDM patients compared with normal pregnant women, and expressed in placental villi. miR-199a-5p can regulate the glucose pathway by inhibiting the expression of methyl CpG-binding protein 2 (MeCP2) and down-regulating canonical transient receptor potential 3 (Trpc3). This suggests that miR-199a-5p may regulate the glucose pathway by regulating methylation levels, leading to the occurrence of GDM.

Indexed as

Diabetes, GestationalMicroRNAsFemaleGlucoseHumansPlacentaPregnancyUp-RegulationGlucoseMicroRNAsmirn199 microRNA, humangestational diabetes mellitusMeCP2MiR-199aplacentaTRPC3

Identifiers

PMID35909537
PMCPMC9330501
OpenAlexW4285388860

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.