Evidence map›Paper›PMID 35879489›Full record

ArticleImmunologic research2022

Somatic hypermutation defects in two adult hyper immunoglobulin M patients.

Hülya Yilmaz, Sinem Fırtına, Merve Sarıtaş, Müge Sayitoğlu, Muhlis Cem Ar

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In one paragraph

Article in Immunologic research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.1field-weighted citation impact, top 60% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Hülya YilmazHematology Department, Faculty of Medicine, Ankara University, Ankara, Turkey. md.hulya.yilmaz@gmail.com.
Sinem FırtınaBioinformatics and Genetics Department, Engineering and Natural Sciences Faculty, Istinye University, Istanbul, Turkey.
Merve SarıtaşInstitute of Graduate Studies in Health Sciences, Istanbul University, Istanbul, Turkey.
Müge SayitoğluAziz Sancar Institute of Experimental Medicine Genetics Department, Istanbul University, Istanbul, Turkey.
Muhlis Cem ArHematology Department, Cerrahpaşa Medical Faculty, Istanbul University-Cerrahpaşa, Istanbul, Turkey.
Istanbul University · TRAnkara University · TRIstanbul University-Cerrahpaşa · TRIstinye University · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hyper immunoglobulin M (HIGM) syndrome is a rare disorder of the immune system with impaired antibody functions. The clinical picture of the patients varies according to the underlying genetic variation. In this study, we identified two novel variants in AID and UNG genes, which are associated with autosomal recessive type HIGM, by targeted next-generation sequencing (NGS) panel. A biallelic 11 base pair deletion (c.278_288delATGTGGCCGAC) in the coding sequence of activation-induced cytidine deaminase (AID) gene was identified in a 36-year-old patient. Biallelic two base pair insertion in exon 7 of uracil nucleoside glycosylase (UNG) gene (c.924_925insGG) was identified in a 40-year-old patient. Both variants were confirmed by Sanger sequencing. HIGM, like many of the other primary immunodeficiencies, is a rare and difficult-to-diagnose entity with heterogeneous clinical phenotypes. It should be suspected in patients with a history of early-onset recurrent respiratory infections, enlarged lymph nodes, and autoimmune disorders. There might be a delay in diagnosis until adulthood especially in subtle cases or if HIGM is not included in the differential diagnosis due lacking of awareness. In this regard, genetic testing with NGS-based diagnostic panels provide a rapid and reasonable tool for the molecular diagnosis of patients with immunodeficiencies and hence, decrease the time to diagnose and prevent infection-related complications associated with increased morbidity and mortality.

Indexed as

Cytidine DeaminaseHyper-IgM Immunodeficiency SyndromeHumansImmunoglobulin MPhenotypeSomatic Hypermutation, ImmunoglobulinCytidine DeaminaseImmunoglobulin MAIDHyper IgM deficiencyPrimary immunodeficiencyUNG

Identifiers

PMID35879489
OpenAlexW4287578705

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.