Evidence map›Paper›PMID 35878875›Full record

ArticleGenes, brain, and behavior2022

Mice lacking proSAAS display alterations in emotion, consummatory behavior and circadian entrainment.

Dipendra K Aryal, Ramona M Rodriguiz, Ngoc Lien Nguyen, Matthew W Pease, Daniel J Morgan, John Pintar, Lloyd D Fricker, William C Wetsel

Open access · hybridAbstract read
In one paragraph

Article in Genes, brain, and behavior, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.8field-weighted citation impact, top 33% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 9 citations in OpenAlex.

  1. Gpr171 regulates embryonic hematopoietic stem cell emergence via ERK1/2 and Notch signaling.Proceedings of the National Academy of Sciences of the United States of America · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 5 institutions in 1 country.

Dipendra K AryalDepartment of Psychiatry and Behavioral Sciences, Duke University Medical Center, Durham, North Carolina, USA.
Ramona M RodriguizDepartment of Psychiatry and Behavioral Sciences, Duke University Medical Center, Durham, North Carolina, USA.
Ngoc Lien NguyenDepartment of Psychiatry and Behavioral Sciences, Duke University Medical Center, Durham, North Carolina, USA.
Matthew W PeaseDepartment of Psychiatry and Behavioral Sciences, Duke University Medical Center, Durham, North Carolina, USA.ORCID 0000-0001-6125-9858
Daniel J MorganDepartment of Anesthesiology and Perioperative Medicine, Pennsylvania State, University College of Medicine, Hershey, Pennsylvania, USA.
John PintarDepartment of Neuroscience and Cell Biology, Rutgers Robert Wood Johnson Medical School, Piscataway, New Jersey, USA.
Lloyd D FrickerDepartments of Molecular Pharmacology and Neuroscience, Albert Einstein College of Medicine, Bronx, New York, USA.
William C WetselDepartment of Psychiatry and Behavioral Sciences, Mouse Behavioral and Neuroendocrine Analysis Core Facility, Duke University Medical Center, Durham, North Carolina, USA.ORCID 0000-0002-3592-7397
Duke Medical Center · USDuke University · USAlbert Einstein College of Medicine · USPennsylvania State University · USRutgers, The State University of New Jersey · US

Funding

Desensitization and downregulation of CB1 during cannabinoid toleranceK01DA037355 · NIDA · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI MORGAN, DANIEL J · 2015 to 2017
$476k
Characterization of a novel JNK-mediated mechanism of cannabinoid toleranceR21DA036385 · NIDA · PENNSYLVANIA STATE UNIV HERSHEY MED CTR · PI MORGAN, DANIEL J · 2013 to 2014
$400k
NIDA NIH HHS K01 DA037355NIDA NIH HHS R21 DA036385
6 · The paper itself

Abstract

ProSAAS is a neuroendocrine protein that is cleaved by neuropeptide-processing enzymes into more than a dozen products including the bigLEN and PEN peptides, which bind and activate the receptors GPR171 and GPR83, respectively. Previous studies have suggested that proSAAS-derived peptides are involved in physiological functions that include body weight regulation, circadian rhythms and anxiety-like behavior. In the present study, we find that proSAAS knockout mice display robust anxiety-like behaviors in the open field, light-dark emergence and elevated zero maze tests. These mutant mice also show a reduction in cued fear and an impairment in fear-potentiated startle, indicating an important role for proSAAS-derived peptides in emotional behaviors. ProSAAS knockout mice exhibit reduced water consumption and urine production relative to wild-type controls. No differences in food consumption and overall energy expenditure were observed between the genotypes. However, the respiratory exchange ratio was elevated in the mutants during the light portion of the light-dark cycle, indicating decreased fat metabolism during this period. While proSAAS knockout mice show normal circadian patterns of activity, even upon long-term exposure to constant darkness, they were unable to shift their circadian clock upon exposure to a light pulse. Taken together, these results show that proSAAS-derived peptides modulate a wide range of behaviors including emotion, metabolism and the regulation of the circadian clock.

Indexed as

AnimalsAnxietyCircadian RhythmConsummatory BehaviorMiceMice, Inbred C57BLMice, KnockoutNeuropeptidesPeptidesReceptors, G-Protein-CoupledGPR171 protein, mouseGpr83 protein, mouseNeuropeptidesPcsk1n protein, mousePeptidesReceptors, G-Protein-Coupledanxietycircadian activityfearmetabolismmiceproSAAS knockout

Identifiers

PMID35878875
PMCPMC9444949
OpenAlexW4287732297

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.